Allicin Improves Lung Injury Induced by Sepsis via Regulation of the Toll-Like Receptor 4 (TLR4)/Myeloid Differentiation Primary Response 88 (MYD88)/Nuclear Factor kappa B (NF-κB) Pathway

机译：大蒜素通过调节Toll样受体4（TLR4）/骨髓分化主要反应88（MYD88）/核因子κB（NF-κB）途径改善败血症诱导的肺损伤

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摘要

BackgroundThe aim of this study was to assess the effects and mechanisms of allicin in a sepsis-induced lung injury in vivo study.

机译：背景本研究的目的是评估大蒜素在败血症诱导的肺损伤体内研究中的作用和机制。

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期刊名称 Medical Science Monitor : International Medical Journal of Experimental and Clinical Research
作者
Ning Shen; Ailing Cheng; Mengru Qiu; Guodong Zang;
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作者单位

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年(卷),期 2019(25),-1
年度 2019
页码 2567–2576
总页数 10
原文格式 PDF
正文语种
中图分类
关键词
Acute Lung Injury Gallic Acid Myeloid Differentiation Factor 88 Receptor Activator of Nuclear Factor-kappa B Toll-Like Receptor 4;

机译：急性肺损伤;没食子酸;髓样分化因子88;核因子-κB受体激活剂;类似Toll的受体4;

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1. NACHT, LRR and PYD domains-containing protein 3 inflammasome is activated and inhibited by berberine via toll-like receptor 4/myeloid differentiation primary response gene 88/nuclear factor-kappa B pathway, in phorbol 12-myristate 13-acetate-induced macrophages [J] . Huang Zhouqing, Ye Bozhi, Han Jibo, Molecular medicine reports . 2018,第2aPta1期

机译：含有含蛋白酶3的含蛋白组蛋白3的Nacht，LRR和Pyd结构域通过Toll样受体4 / myeloid分化初级反应基因88 /核因子-Kappa B途径激活和抑制肺栓塞13-醋酸盐诱导的巨噬细胞
2. Hypoxia-Inducible Ubiquitin Specific Peptidase 13 Contributes to Tumor Growth and Metastasis via Enhancing the Toll-Like Receptor 4/Myeloid Differentiation Primary Response Gene 88/Nuclear Factor-κB Pathway in Hepatocellular Carcinoma [J] . Gao Shan, Chen Tianxiang, Li Lijie, Frontiers in Cell and Developmental Biology . 2020,第1期

机译：缺氧诱导的泛素特异性肽酶13通过增强肝细胞癌中的收缩受体4 / myeloid分化初级反应基因88 /核因子-κB途径有助于肿瘤生长和转移
3. Propofol Potentiates Sevoflurane-Induced Inhibition of Nuclear Factor-kappa B-Mediated Inflammatory Responses and Regulation of Mitogen-Activated Protein Kinases Pathways via Toll-like Receptor 4 Signaling in Lipopolysaccharide-Induced Acute Lung Injury in Mice [J] . Liu Wei, Zhu Honghua, Fang Hao The American Journal of the Medical Sciences . 2017,第5期

机译：异丙酚增强了七氟醚诱导的核因子-κB介导的抑制核因子-Kappa介导的炎症反应和通过Toll样受体4信号传导在小鼠血液多糖诱导的小鼠中的急性肺损伤的信号传导
4. Granulocyte colony -stimulating factor induces egr-1 up-regulation in myeloid cells through a MEK/ERK-dependent pathway and through interaction of serum response element -binding proteins [D] . Mora-Garcia, Patricia 2002

机译：粒细胞集落刺激因子通过MEK / ERK依赖性途径以及血清反应因子结合蛋白的相互作用诱导髓样细胞中的egr-1上调。
5. Protective Effect of Hesperidin Against Sepsis-Induced Lung Injury by Inducing the Heat-Stable Protein 70 (Hsp70)/Toll-Like Receptor 4 (TLR4)/ Myeloid Differentiation Primary Response 88 (MyD88) Pathway [O] . Xiaoyan Yuan, Jinyuan Zhu, Qi Kang, 2019

机译：橙皮苷通过诱导热稳定蛋白70（Hsp70）/ Toll样受体4（TLR4）/髓样分化主要反应88（MyD88）途径对败血症所致肺损伤的保护作用
6. Allicin Improves Lung Injury Induced by Sepsis via Regulation of the Toll-Like Receptor 4 (TLR4)/Myeloid Differentiation Primary Response 88 (MYD88)/Nuclear Factor kappa B (NF-κB) Pathway [O] . Ning Shen, Ailing Cheng, Mengru Qiu, 2019

机译：Allicin通过调节Toll样受体4（TLR4）/ myeloid分化初级反应88（MyD88）/核因子Kappa B（NF-κB）途径（NF-κB）途径（TLR4）/核因子kappa（NF-κB）途径（TLR4）诱导肺损伤

Allicin Improves Lung Injury Induced by Sepsis via Regulation of the Toll-Like Receptor 4 (TLR4)/Myeloid Differentiation Primary Response 88 (MYD88)/Nuclear Factor kappa B (NF-κB) Pathway

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