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A quantitative histopathological study of right bundle branch block complicating acute anteroseptal myocardial infarction.

机译:右束支传导阻滞并发急性前房间隔心肌梗死的定量组织病理学研究。

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摘要

The aim of the present study was to evaluate whether necrosis of the right bundle branch is responsible for development of right bundle branch block in acute myocardial infarction. Twenty patients with acute anteroseptal myocardial infarction were studied--10 with right bundle branch block (group A) and 10 without (group B)--to evaluate by serial sectioning the pathological extent of myocardial infarction surrounding the right bundle branch and also that of right bundle branch necrosis. Myocardial infarction reached the right bundle branch more than 8 mm above the moderator band in all of group A, whereas myocardial infarction reached the right bundle branch less than 3 mm above the moderator band in only three patients in group B. Nine hearts in group A showed significant necrosis of the right bundle branch. In group B and in one case with transient right bundle branch block no necrosis was found. The occurrence of right bundle branch block was almost entirely explained by necrosis of the right bundle branch, but transient right bundle branch block did develop without necrosis of the right bundle branch.
机译:本研究的目的是评估急性心肌梗死中右束支的坏死是否与右束支传导阻滞有关。研究了20例急性前房间隔心肌梗塞患者--10右束支传导阻滞(A组)和10例不伴右束支传导阻滞(B组)-通过连续切片评估右束支周围的心肌梗死的病理范围以及右束支坏死。在所有A组中,心肌梗死均超过调节带以上8 mm,而在B组中只有3位患者达到右束分支在右侧3mm以上。显示右束支明显坏死。在B组中,在一种情况下伴有短暂性右束支传导阻滞,未发现坏死。右束支的坏死几乎可以完全解释右束支传导阻滞的发生,但是在没有右束支坏死的情况下,瞬时性右束支传导阻滞的确发展了。

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