首页> 外文学位 >Age -dependent induction of matrix metalloproteinase-1 is a redox -sensitive process requiring the activation of the c-Jun N -terminal kinase pathway

【24h】

Age -dependent induction of matrix metalloproteinase-1 is a redox -sensitive process requiring the activation of the c-Jun N -terminal kinase pathway

机译：年龄依赖性诱导基质金属蛋白酶-1是氧化还原敏感的过程，需要激活c-Jun N末端激酶途径

获取原文

获取原文并翻译 | 示例

页面导航

著录项
相似文献
相关主题

著录项

作者
Dasgupta, Jaya;
展开▼
作者单位

Albany Medical College;

展开▼
授予单位 Albany Medical College;
学科
学位 Ph.D.
年度 2008
页码 185 p.
总页数 185
原文格式 PDF
正文语种 eng
中图分类
关键词
入库时间 2022-08-17 11:57:03

相似文献

外文文献
中文文献
专利

1. Activation of p38 and N-acetylcysteine-sensitive c-Jun NH2-terminal kinase signaling cascades is required for induction of apoptosis in Parkinson's disease cybrids [J] . Isaac G. Onyango, Jeremy B. Tuttle, James P. Bennett Jr. Molecular and cellular neurosciences . 2005,第3期

机译：需要激活p38和N-乙酰半胱氨酸敏感的c-Jun NH2-末端激酶信号级联反应来诱导帕金森氏病细胞凋亡
2. c-Jun N-terminal kinase binding domain-dependent phosphorylation of mitogen-activated protein kinase kinase 4 and mitogen-activated protein kinase kinase 7 and balancing cross-talk between c-Jun N-terminal kinase and extracellular signal-regulated ki [J] . Repici M, Mare L, Colombo A, Neuroscience: An International Journal under the Editorial Direction of IBRO . 2009,第1期

机译：c-Jun N末端激酶结合域依赖的丝裂原活化蛋白激酶4和7的磷酸化以及c-Jun N末端激酶与细胞外信号调节ki之间的串扰平衡
3. Compartment-specific regulation of extracellular signal-regulated kinase (ERK) and c-Jun N-terminal kinase (JNK) mitogen-activated protein kinases (MAPKs) by ERK-dependent and non-ERK-dependent inductions of MAPK phosphatase (MKP)-3 and MKP-1 in diff [J] . Reffas S., Schlegel W. The Biochemical Journal . 2000,第Pta3期

机译：通过ERK依赖性和非ERK依赖性MAPK磷酸酶（MKP）诱导，对细胞外信号调节激酶（ERK）和c-Jun N端激酶（JNK）丝裂原激活的蛋白激酶（MAPK）进行室特异性调节3和MKP-1在差异
4. Particulate Joint Replacement Materials Induce Apoptosis of Rabbit Synoviocytes Cell Line HIG-82 through c-Jun N-Terminal Kinase (JNK) Pathway [C] . Chu Feng-Min, Lian Shuang-Shii, Sung Yen-Jen International Conference on Biomedical Engineering and Informatics;BMEI '09 . 2009

机译：颗粒关节替代材料通过c-Jun N末端激酶（JNK）途径诱导兔滑膜细胞HIG-82细胞凋亡
5. Roles of p53, p21waf1 and the phosphoinositide 3-kinase pathway in regulation of c-Jun amino terminal kinase [D] . Xue, Yue. 2004

机译：p53，p21waf1和磷酸肌醇3激酶途径在c-Jun氨基末端激酶调节中的作用
6. Tumour necrosis factor-induced activation of c-Jun N-terminal kinase is sensitive to caspase-dependent modulation while activation of mitogen-activated protein kinase (MAPK) or p38 MAPK is not. [O] . Ahmed A A Mohamed, Orla J Jupp, Helen M Anderson, 2002

机译：肿瘤坏死因子诱导的c-Jun N末端激酶激活对caspase依赖性调节敏感而丝裂原激活的蛋白激酶（MAPK）或p38 MAPK激活则不敏感。
7. Activation of the Hematopoietic Progenitor Kinase-1 (HPK1)-dependent, Stress-activated c-Jun N-terminal Kinase (JNK) Pathway by Transforming Growth Factor β (TGF-β)-activated Kinase (TAK1), a Kinase Mediator of TGF β Signal Transduction [O] . Wenfu Wang, Guisheng Zhou, Mickey C.-T. Hu, 1997

机译：通过转化生长因子β（TGF-β） - TGF的激酶介体，通过转化生长因子β（TGF-β），通过TGF的激酶介质来激活造血祖酶-1（HPK1） - 依赖性，应激活化的C-JUM N-末端激酶（JNK）途径β信号转导

Age -dependent induction of matrix metalloproteinase-1 is a redox -sensitive process requiring the activation of the c-Jun N -terminal kinase pathway

著录项

相似文献

相关主题

期刊订阅