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Trace mineral assessment in practice

机译:追踪矿物评估在实践中

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Providing proper trace mineral nutrition in animals involves an important balancing act. Many factors must be reconciled to be sure that the essential needs for optimal production and performance are met without upsetting the balances of other nutritional elements or poisoning the animal. Those factors include other nutritional constituents, species and production class of the animal to be fed, type of feed involved, and essential requirements of the trace mineral involved. As with any clinical case, assessment of the mineral status of an animal or herd involves inputs from a variety of sources, including clinical history, clinical signs, clinical pathology, pathology (if animals have been lost), analytical chemistry, and bioassay. Unfortunately,no one source alone will provide the entire answer to a problem. This is especially true with trace mineral imbalances since deficiencies (in particular) are often manifested in terms of increased incidence of other, possibly common, diseases. Historically, a trace mineral deficiency may cause an outbreak of nutritional disease (e.g. white muscle disease in horses or cattle), or may be disguised by other concurrent diseases. The latter occurs with more subtle deficiencies of minerals such as selenium, copper, and zinc especially since those minerals are closely tied to proper immune function. Poisoning often is suggested historically when a feed or environmental change coincides with the onset of specific disease. The onset of disease could be delayedor happen after a period of accumulation in some cases, such as with the buildup of copper, followed by sudden and catastrophic losses of animals. In some instances, such as when molybdenum is present in excess, the signs of disease may be a deficiency of another metal, copper. When investigating a possible trace mineral problem, one may identify errors in supplementation, that may lead to either undersupplementation and deficiency, or oversupplementation and toxicosis. Overdosing of animals with a given element(s) have resulted from errors at all stages of formulation, mixing, and delivery. Misguided articles have encouraged farmers to add extra mineral to some rations as sort of the old "more is always better" attitude with disastrous results. Even if the ration is properly prescribed by the rancher or the nutritionist, transcription errors may lead to improper formulas leading to either toxicosis or deficiency. Once prescribed, it is then incumbent on the mill to properly mix the material, which hopefully arrived at the mill in the proper levels in the first place. Once mixed, a ration must be properly labeled. If not a total mixed ration, then those on the ranch must be sure that the total ration is balanced for each mineral as they provide the feed. Important decisions made at that level may include: How to best supplement? Which route to use? Is the feed best? How long will the product persist? Is overdosage possible? And other questions.
机译:在动物提供适当的微量元素的营养涉及到一个重要的平衡作用。许多因素必须加以协调,以确保最佳的生产和性能的基本需求没有打乱其他营养元素的平衡或中毒的动物见面。这些因素包括其它营养成分,种类和生产类动物的要被馈送,涉及进料的类型以及所涉及的微量矿物质的基本要求。正如任何临床情况下,动物或畜群的矿物质状况评估涉及从多种来源,包括临床病史,临床症状,临床病理学,病理(如果动物已经丢失),分析化学,生物测定和投入。不幸的是,没有单独一个源将提供一个完整的答案的一个问题。因为缺陷(尤其是)在增加的其他可能常见,疾病发病的术语往往表现这与微量矿物质失衡尤其如此。在历史上,微量矿物质缺乏可能会导致营养性疾病(在马或牛例如白色肌肉疾病)的爆发,或者可以通过其他并发疾病被伪装。后者发生与矿物质的更细微的缺陷,诸如硒,铜和锌特别是因为这些矿物是紧密联系在一起适当的免疫功能。中毒常常被认为历史上,当饲料或环境变化恰逢特定疾病的发作。疾病的发作可能是在某些情况下,如铜的积累,其次是动物的突然和灾难性损失一段时间的积累后delayedor发生。在一些情况下,诸如当钼过量存在,疾病的符号可以是另一种金属的缺乏,铜。当调查可能微量元素的问题,一个可识别的错误的补充,可能导致要么undersupplementation和不足,或oversupplementation和中毒。动物与给定的元件(一个或多个)的过量在配制,混合和输送的各个阶段导致从错误。被误导的文章都鼓励农民额外的矿物质一些口粮添加为某种带来灾难性的后果老“更多的是总是更好”的态度。即使比正常由农场主或营养师规定,抄写错误可能会导致错误的公式,导致无论是中毒或不足。一旦规定,它是那么话茬磨正确地混合材料,它首先希望抵达工厂在适当的水平。一旦混合,配给必须正确标注。如果不是全混合日粮,那么那些在农场必须确保总日粮对每个矿物平衡,因为它们提供的饲料。在这一水平做出了重要的决定可能包括:如何最好的补充?哪条路线使用?是饲料最好?该产品将持续多久?是过量可能吗?和其他问题。

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