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出血坏死性胰腺炎

出血坏死性胰腺炎的相关文献在1990年到2017年内共计75篇,主要集中在外科学、内科学、临床医学 等领域,其中期刊论文75篇、专利文献11416篇;相关期刊58种,包括中国社区医师、内蒙古中医药、现代临床医学等; 出血坏死性胰腺炎的相关文献由148位作者贡献,包括唐治奎、张莉、李华等。

出血坏死性胰腺炎—发文量

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论文:75 占比:0.65%

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论文:11416 占比:99.35%

总计:11491篇

出血坏死性胰腺炎—发文趋势图

出血坏死性胰腺炎

-研究学者

  • 唐治奎
  • 张莉
  • 李华
  • 李德荣
  • 翁桂花
  • 丁依玲
  • 乔凡丁
  • 于艳
  • 于骅
  • 期刊论文
  • 专利文献

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    • 易冬梅
    • 摘要: 目的:通过病情观察及护理提高出血坏死性胰腺炎的有效率.方法:观察51例急性出血坏死性胰腺炎患者的病情并进行护理.密切观察病人生命体征及腹痛情况,加强各种管道护理、营养支持及并发症的观察与护理.结果:治疗后观察组总有效率明显高于对照组,两组比较χ2=3.71,P<0.05,差异显著;观察组总有效率高达88.88%,明显高于对照组有效率66.66%.结论:严密观察病情和精心护理对提高治疗效果、改善患者预后有重要意义.
    • 高迪; 黄澜; 张欣
    • 摘要: 目的 探讨不同的鼻饲营养途径对出血坏死性胰腺炎患者疾病发展状况的影响.方法 选择162例连续禁食、胃肠减压48 h以上,且肠蠕动恢复、腹痛已缓解的出血坏死性胰腺炎患者按入院时间先后分成两组,对照组行鼻胃管鼻饲(70例),观察组鼻空肠管鼻饲(92例).记录两组患者的并发症发生情况、淀粉酶恢复正常时间、白蛋白恢复正常时间、C反应蛋白恢复正常时间以及住院时间.结果 观察组患者在出现感染、消化道出血、腹泻、腹腔脓肿、多脏器功能不全及死亡例数均显著少于对照组,差异有统计学意义(P<0.05).观察组患者的淀粉酶恢复正常时间、白蛋白恢复正常时间、C反应蛋白恢复正常时间、住院时间均显著短于对照组,差异有统计学意义(P<0.05).结论 对出血坏死性胰腺炎患者护理中使用螺旋空肠管进行肠内营养优势明显,并发症显著减少,恢复时间短,护理难度低,值得临床推广.
    • 朱美凤
    • 摘要: 目的:分析急性胰腺炎的CT表现,提高诊断水平.方法:回顾分析临床确诊的30例急性胰腺炎患者的螺旋CT表现,进行评价.结果:其中25例为急性水肿性胰腺炎,5例为出血坏死性胰腺炎,均经临床、实验室检查及腹腔穿刺证实.结论:螺旋CT对急性胰腺炎显示率达98%,不仅可确定诊断,而且能判断炎症的范围、程度及并发症,还可在CT引导下穿刺、抽液、引流,是目前诊断急性胰腺炎的主要方法.
    • 楚丽雅
    • 摘要: @@%急性出血坏死性胰腺炎是病死率较高一种急腹症,其治疗理念随医学发展而转变,护理内容须随治疗理念转变而不断完善,整体护理对该病疗效的提高有明显的促进作用.
    • 伍彩虹; 靳蕾
    • 摘要: 目的:总结重症胰腺炎术后的基础护理和专科护理方法,提高护理质量,促进病人的恢复.方法:对6例重症胰腺炎术后护理进行分析.结果:出现呼吸窘迫1例,休克1例,胰漏2例.1例转上级医院,5例痊愈出院.结论:积极加强术后的基础护理和专科护理能提高手术成功率缩短住院天数.
    • 刘林; 于骅; 钦琦; 张军港; 刘洋; 邓世昌; 赵刚; 王春友
    • 摘要: Objective To investigate the effects of intestinal ischemia reperfusion (IIR) on the progression of inflammatory reaction in hemorrhagic necrosis pancreatitis (HNP).Methods Eighty rats were randomly divided into sham operation (SO) group,acute edematous pancreatitis (AEP) group,AEP + IIR group and HNP group according to the random number table.Erythrocyte velocity (EV),functional capillary density (FCD) and leukocyte adherence (LA) were observed at 0,1,2,3 and 6 hours after the models were completed.The serum levels of tumor necrosis factor-α (TNF-α) and interleukin-6 (IL-6) were detected.All data were analyzed by using the analysis of variance or the t test.Results The level of EV in the AEP group significantly decreased at 1 hour,and got increased at 3 hours,while the level of EV in the AEP group was still significantly lower than that in the SO group ( t =9.60,P < 0.05 ).The levels of EV in the AEP + IIR group and HNP group constantly decreased,and increased at 6 hours,but were continually lower than that in the AEP group ( t =6.03,6.12,P <0.05 ).The level of FCD in the AEP group was significantly lower than that in the SO group at 3 hours ( t =8.20,P<0.05).The levels of FCD in the AEP + IIR group and HNP group were significantly lower than that in the AEP group at 3 hours (t =35.60,23.80,P < 0.05 ).Compared with AEP group,the level of LA in the AEP group was significantly increased at 1 hour ( t =75.00,P < 0.05 ) and reached peak at 3 hours.The levels of LA in the AEP + IIR group and HNP group were significantly higher than that in the AEP group at 1,2,3,6 hours (t =23.00,29.50,53.00,38.70,23.10,48.20,39.20,47.50,P<0.05).Compared with SO group,the level of TNF-α in the AEP group significantly increased since l hour (t =77.00,P < 0.05),and began to decrease at 3 hours; the levels of TNF-α in the AEP +IIR group and HNP group at 2 hours were significantly higher than that in the AEP group (t =23.50,18.10,P<0.05).The levels of IL-6 in the AEP group at 1,2,3,6 hours were significantly higher than those in the SO group ( t =93.50,146.00,243.60,209.20,P < 0.05 ).The levels of IL-6 in the AEP + IIR group and HNP group at 1 hour were not significantly different from that in the AEP group ( t =2.30,2.03,P > 0.05),while the levels of IL-6 in the AEP + IIR group and HNP group at 2 hours were significantly higher than that in the AEP group (t =35.63,29.80,P < 0.05 ).Conclusion IIR may enhance the inflammatory reaction of AEP and IIR might be one of the factors to exaggerate the inflammatory reaction of HNP.%目的 探讨肠道缺血再灌注(IIR)在出血坏死性胰腺炎(HNP)早期炎性反应中的作用.方法 根据随机数字表法将80只大鼠分为4组,每组20只:假手术组(SO组),急性水肿型胰腺炎组(AEP组),急性水肿型胰腺炎合并肠道缺血再灌注组(AEP+ IIR组)和出血坏死性胰腺炎组(HNP组).于建立模型后0、1、2、3、6h分别检测小肠系膜微循环红细胞流速(EV)、功能毛细血管密度(FCD)及白细胞黏附数(LA),并测定血清TNF-α及IL-6水平.多组比较采用方差分析,两两比较采用t检验.结果 AEP组EV在1h明显下降,在3h升高,但仍显著低于SO组(t=9.60,P<0.05);而AEP+ IIR组及HNP组EV则持续下降,直至6h升高,但仍显著低于AEP组(t=6.03,6.12,P<0.05).AEP组FCD在3h时显著低于SO组(t=8.20,P<0.05);而AEP+ IIR组及HNP组FCD在3h后显著低于AEP组(t=35.60,23.80,P<0.05).AEP组LA在1h较SD组明显升高(t=75.00,P<0.05),在3h达到峰值;而AEP+ IIR组及HNP组LA在1、2、3、6h均显著高于AEP组(t=23.00,29.50,53.00,38.70,23.10,48.20,39.20,47.50,P<0.05).与SO组比较,AEP组TNF-α在1h显著升高(t=77.00,P<0.05),3h后逐渐下降,而AEP+ IIR组及HNP组TNF-α在2h后显著高于AEP组(t=23.50,18.10,P<0.05).AEP组IL-6水平在1、2、3、6h持续高于SO组(t=93.50,146.00,243.60,209.20,P<0.05),而AEP+ IIR组及HNP组IL-6在1h与AEP组比较,差异无统计学意义(t =2.30,2.03,P>0.05),但在2h后显著高于AEP组(t=35.63,29.80,P<0.05).结论 IIR可进一步加重AEP早期炎性反应,提示IIR是HNP早期发生、发展的重要促进因素.
    • 刘富瑶; 崔乃强
    • 摘要: 对于急性重症坏死性胰腺炎的治疗,除了坚持胰腺炎治疗的一般性原则外,还应针对胰腺炎的特殊病情选用其独特的治疗方法,并把握住治疗的恰当时机,对期间出现的失血性休克症状进行分析与积极救治.
    • 翁桂花
    • 摘要: 目的:对出血坏死性胰腺炎患者进行临床观察,探讨血坏死性胰腺炎临床特点及护理措施,总结护理经验.方法:2002~2009年收治出血坏死性胰腺炎患者25例,观察病情及护理情况.结果:本组25例患者,非手术治疗16例,全部治愈,均无并发症.手术治疗治疗9例,死亡1例,因术后并发重度出血性休克经抢救无效而死亡.结论:加强对出血坏死性胰腺炎患者的临床观察,采取有效的护理措施,争取治疗时间.
    • 崔爱芝
    • 摘要: 出血坏死性胰腺炎病情凶险、病程长,多因胆道疾患、酒精中毒、暴饮暴食及外伤等原因引起。发作时疼痛剧烈而持续,呈绞痛、刀割样痛,一般镇痛剂难以缓解,可并发休克、出血、电解质紊乱、多器官功能衰竭而死亡”⑴。医护人员密切酝合、快诊速治、周密护理对挽救患者的生命有着极为重要的意义。2009年12月~2010年6月,我们共收治出血坏死性胰腺炎患者18例,经积极治疗与精心护理,取得满意效果。现将围术期护理体会报告如下。
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