The process in order to increase the electrical resistance of some metals, and in particular, those which are adapted to constitute the body of the light for incandescent lamps

• 1. 用里奥塞尔模塑体从含重金属的介质中除去重金属的方法以及含有所吸附重金属的纤维素模塑体与其应用 [P] . 中国专利： CN1547499A . 2004-11-17
• 2. 一种能够最小程度增加硅片体金属的退火炉装置及其方法 [P] . 中国专利： CN113883895A . 2022-01-04
• 3. A procedure to increase the electrical resistance of incandescent bodies Made Of Metal Melt at high temperatures. [P] . ES38100A1 . 1906-06-01

  机译： 在高温下增加由金属制成的白炽体的电阻的过程。

• 4. Electric incandescent lamp illuminating the body of which is constituted by the tantalum carbide, or of a metal of the same group and method of manufacture of said lamp. [P] . 法国专利： FR755026A . 1933-11-18

  机译： 照明其主体的电白炽灯由碳化钽或相同种类的金属构成，并制造所述灯。

• 5. Electrical incandescent lamp has light body(ies) in lamp vessel with flat, esp. strip-shaped, light body element(s) radiating in infrared and visible regions, infrared reflective filter(s) [P] . 德国专利： DE19843852A1 . 2000-03-30

  机译： 白炽灯在带有扁平，特别是esp的灯泡中具有发光体。在红外和可见光区域辐射的带状光体元件，红外反射滤光片

• 1. THE INVESTIGATION OF THE METAL DAMAGE AT THE CREEP BY THE METHOD OF ELECTRICAL RESISTANCE MEASURING [J] . 力学学报：英文版 . 1991,第002期
• 2. Pullet Housing Comparison of Dimmable Incandescent Lamps vs. Incandescent Flexitube Lite vs. Compact Fluorescent Light Systems [C] . Ron MacDonald, Katie Gibb, Mike Kazmaier, Annual International Meeting of the American Society of Agricultural and Biological Engineers . 2009
• 3. Wear resistance and electrical property of infrared processed copper/tungsten carbide composites. [D] . Deshpande, Pranav K. 2006
• 4. Comparative Analysis of Lighting Characteristics and Ultraviolet Emissions from Commercial Compact Fluorescent and Incandescent Lamps [O] . Mahtab Azizi, Rostam Golmohammadi, Mohsen Aliabadi 2016
• 5. The Value of Incandescent Lamp in Terms of Lighting Culture(130Years of History and the Future of the Incandescent Lamp) [O] . Kaoru Mende 2010

• 1. BACKGROUND： Vibrio vulnifi cus inside the body could activate the NF-!B signaling pathwayand initiate the inflammatory cascade. The lung is one of the earliest organs affected by sepsisassociated with acute lung injury. High mobility group protein B1 （HMGB1） is an important late-actingpro-infl ammatory cytokine involving in the pathophysiology of sepsis. It is also involved in the injuryprocess in the lung, liver and intestine. There has been no report on the involvement of HMGB1 inVibrio vulnifi cus sepsis-induced lung injury.METHODS： Sixty rats were randomly divided into a normal control group （group A, n=10） anda Vibrio vulnificus sepsis group （group B, n=50）. Sepsis was induced in the rats by subcutaneousinjection of Vibrio vulnificus （concentration 6×108 cfu/mL, volume 0.1 mL/100g）） into the left lowerlimbs. The rats in group B were sacrifi ced separately 1, 6, 12, 24, and 48 hours after the infection.Their lungs were stored as specimens, lung water content was measured, and lung pathology wasobserved under a light microscope. The expressions of the HMGB1 gene and protein in the lungswere detected by RT-PCR and Western blot. Data were analyzed with one-way analysis of variance（ANOVA） and the LSD method for pair-wise comparison between the two groups. P〈0.05 wasconsidered statistically signifi cant.RESULTS： Compared to group A （0.652±0.177）, HMGB1 mRNA expression in the lungs ofgroup B was signifi cantly higher at 0 hour （1.161±0.358, P=0.013）, 24 hours （1.679±0.235, P=0.000）,and 48 hours （1.258±0.274, P=0.004） （P〈0.05）, and peaked at 24 hours. Compared to group A（0.594±0.190）, HMGB1 protein expression at 6 hours （1.408±0.567, P=0.026） after infection wassignificantly increased （P〈0. 05）, and peaked at 24 hours （2.415±1.064, P=0.000） after infection.Compared to group A （0.699±0.054）, lung water content was significantly increased at 6 hours（0.759±0.030, P=0.001）,12 hours （0.767±0.023, P=0.000）, 24 hours （0.771±0.043, P=0.000） and 48hours （0.789±0.137, P=0.000） after infection （P〈0.05）. Compared to group A, pathological changesat 12 hours in group B indicate marked pulmonary vascular congestion, interstitial edema andinfl ammatory infi ltration. Alveolar cavity collapse and boundaries of the alveolar septum could not beclearly identifi ed.CONCLUSION： Vibrio vulnifi cus sepsis can lead to injury in rat lungs, and increased HMGB1expression in lung tissue may be one of the mechanisms for injury from Vibrio vulnifi cus sepsis. [J] . Qiao-meng Qiu ,Zhong-wang Li ,Lu-ming Tang . 世界急诊医学杂志(英文) . 2011,第4期
• 2. Electrical Stimulation Greatly Increases Settlement, Growth, Survival, and Stress Resistance of Marine Organisms [J] . Thomas J. Goreau . 自然资源（英文） . 2014,第10期
• 3. Effect of retained austenite and nonmetallic inclusions on the thermal/electrical properties and resistance spot welding nuggets of Si-containing TRIP steels [J] . V.H. Vargas ,I. Mejía ,V.H. Baltazar-Hernández . 矿物冶金与材料学报 . 2019,第001期
• 4. Electrical Resistance Measurement of Glass Transition and Crystallization Characteristics of Zr-Al-Cu-Ni Metallic Glasses [J] . Yaping WANG and Ke LUState Key Laboratory for RSA Institute of Metal Research Chinese Academy of Sciences Shenyang 110016 China . 材料科学技术：英文版 . 2002,第6期
• 5. 具有金属内衬复合材料缠绕结构成型和使用过程多场耦合一体化分析 [C] . 陈浩然 ,任明法 . 2007年全国复合材料力学研讨会 . 2007
• 6. 铁磁金属-金属/绝缘体颗粒膜中的相分离、晶化过程和巨磁电阻 [A] . 周宁 . 2003