Method of extracting lipides from waste animal tissue remaining after a hydrolysis process

• 1. 从海洋的和水生的动物组织中提取脂质的方法 [P] . 中国专利： CN1269940C . 2006.08.16
• 2. 从海洋的和水生的动物组织中提取脂质的方法 [P] . 中国专利： CN1324394A . 2001-11-28
• 3. METHOD OF EXTRACTING LIPIDES FROM WASTE ANIMAL TISSUE REMAINING AFTER A HYDROLYSIS PROCESS [P] . PL342983A1 . 2002-04-08

  机译： 水解后从残留的动物组织中提取脂质的方法

• 4. METHOD OF OBTAINING LIPIDE-CONTAINING RAW MATERIALS FROM WASTE NERVE TISSUE OF SLAUGHTER ANIMALS AFTER SEPARATING CHOLESTERIN THEREFROM [P] . PL114690B2 . 1981-02-28

  机译： 分离胆甾醇后从屠宰动物废物神经组织中获取含脂原料的方法

• 5. METHOD OF OBTAINING LIPIDE-CONTAINING RAW MATERIALS FROM WASTE NERVE TISSUE OF SLAUGHTER ANIMALS AFTER SEPARATING CHOLESTERIN THEREFROM [P] . PL207034A2 . 1980-01-28

  机译： 分离胆甾醇后从屠宰动物废物神经组织中获取含脂原料的方法

• 1. Toxicological studies for some agricultural waste extracts on mosquito larvae and experimental animals [J] . Somia El-Maghraby, Galal A Nawwar, Reda FA Bakr, 亚太热带生物医学杂志（英文版） . 2012,第007期
• 2. Toxicological studies for some agricultural waste extracts on mosquito larvae and experimental animals [J] . Somia El-Maghraby, Galal A Nawwar, Reda FA Bakr, 亚太热带生物医学杂志：英文版 . 2012,第007期
• 3. Optimization of process conditions for infected animal tissues by alkaline hydrolysis technology [C] . Tao Wang, Jin-hui Wu, Ying Yi International Conference on Waste Management and Technology . 2017
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• 5. Effects of the Salt-Processing Method on the Pharmacokinetics and Tissue Distribution of Orally Administered Morinda officinalis How. Extract [O] . Ji Shi, Xiaohang Ren, Jia Wang, 2020
• 6. Extracting and physicochemical properties of carotenoprotein from shrimp processing waste by proteases-mediated hydrolysis [O] . Parastoo Pourashouri, Hojat Mirsadeghi, Aynaz Khodanazary 2021

• 1. Anticorrosive properties of extracts from waste of rice processing [J] . . 材料研究与应用 . 2005,第002期
• 2. Anticorrosive properties of extracts from waste of rice processing [J] . Kharchenko ,U. ,V. . 材料研究与应用 . 2005,第003期
• 3. BACKGROUND： Vibrio vulnifi cus inside the body could activate the NF-!B signaling pathwayand initiate the inflammatory cascade. The lung is one of the earliest organs affected by sepsisassociated with acute lung injury. High mobility group protein B1 （HMGB1） is an important late-actingpro-infl ammatory cytokine involving in the pathophysiology of sepsis. It is also involved in the injuryprocess in the lung, liver and intestine. There has been no report on the involvement of HMGB1 inVibrio vulnifi cus sepsis-induced lung injury.METHODS： Sixty rats were randomly divided into a normal control group （group A, n=10） anda Vibrio vulnificus sepsis group （group B, n=50）. Sepsis was induced in the rats by subcutaneousinjection of Vibrio vulnificus （concentration 6×108 cfu/mL, volume 0.1 mL/100g）） into the left lowerlimbs. The rats in group B were sacrifi ced separately 1, 6, 12, 24, and 48 hours after the infection.Their lungs were stored as specimens, lung water content was measured, and lung pathology wasobserved under a light microscope. The expressions of the HMGB1 gene and protein in the lungswere detected by RT-PCR and Western blot. Data were analyzed with one-way analysis of variance（ANOVA） and the LSD method for pair-wise comparison between the two groups. P〈0.05 wasconsidered statistically signifi cant.RESULTS： Compared to group A （0.652±0.177）, HMGB1 mRNA expression in the lungs ofgroup B was signifi cantly higher at 0 hour （1.161±0.358, P=0.013）, 24 hours （1.679±0.235, P=0.000）,and 48 hours （1.258±0.274, P=0.004） （P〈0.05）, and peaked at 24 hours. Compared to group A（0.594±0.190）, HMGB1 protein expression at 6 hours （1.408±0.567, P=0.026） after infection wassignificantly increased （P〈0. 05）, and peaked at 24 hours （2.415±1.064, P=0.000） after infection.Compared to group A （0.699±0.054）, lung water content was significantly increased at 6 hours（0.759±0.030, P=0.001）,12 hours （0.767±0.023, P=0.000）, 24 hours （0.771±0.043, P=0.000） and 48hours （0.789±0.137, P=0.000） after infection （P〈0.05）. Compared to group A, pathological changesat 12 hours in group B indicate marked pulmonary vascular congestion, interstitial edema andinfl ammatory infi ltration. Alveolar cavity collapse and boundaries of the alveolar septum could not beclearly identifi ed.CONCLUSION： Vibrio vulnifi cus sepsis can lead to injury in rat lungs, and increased HMGB1expression in lung tissue may be one of the mechanisms for injury from Vibrio vulnifi cus sepsis. [J] . Qiao-meng Qiu ,Zhong-wang Li ,Lu-ming Tang . 世界急诊医学杂志(英文) . 2011,第4期
• 4. LC-MS-MS法测定动物组织中苯乙醇胺A残留的方法研究 [C] . LI Shu-gang ,李树纲 ,ZHUO MA Cai-rang . 2016第六届中国兽药大会（中国畜牧兽医学会动物药品学分会第五届全国会员代表大会暨2016学术年会、中国微生物学会兽医微生物学专业委员会、中国畜牧兽医学会生物制品学分会2016年联合学术大会） . 2016
• 5. 基于脂质组学条件优化的猪肉中β--受体激动剂残留分析方法研究 [A] . 张琳 . 2020