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Total knee arthroplasty for neuropathic joint disease after severe bone destruction eroded the tibial tuberosity

机译:严重骨破坏侵蚀胫骨结节后的全膝关节置换术治疗神经性关节疾病

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This article describes a patient in whom total knee arthroplasty was performed for neuropathic joint disease secondary to diabetes mellitus after severe bone destruction eroded the tibial tuberosity. At initial examination, radiographs of the knee showed bone destruction in the medial and anterior regions of the tibia, and fine bone fragments were seen in the joint. Conservative therapy was performed using a brace. However, bone destruction gradually advanced, and 10 months after the initial examination, radiographs of the knee showed bone destruction in the lateral condyle of the femur and advanced bone destruction of the anterior tibia; the tibial tuberosity was missing. It is rare for the tibial tuberosity in the anterior tibia to disappear. If this happens, reconstruction is difficult and total knee arthroplasty becomes complicated. For the bone defect in the tibia, cement was used to recreate the shape of the anterior surface of the tibia. It was possible to minimize the volume of bone resection and morphologically reconstruct the tibial tuberosity. The patient recovered quickly. At postoperative week 5, the patient was able to walk using a cane. Thirty-six months after total knee arthroplasty, knee extension was 0°, flexion was 120°, extension lag was 5°, knee score improved from 40 points to 94 points, and functional score improved from 20 points to 75 points. However, long-term implant stability needs to be carefully monitored.
机译:本文介绍了在严重的骨骼破坏侵蚀胫骨结节后,对因糖尿病继发的神经性关节疾病进行全膝关节置换术的患者。初次检查时,膝部X射线照片显示胫骨内侧和前部的骨质破坏,并且在关节处看到细小的骨碎片。使用支架进行保守治疗。然而,骨破坏逐渐发展,在初步检查后的10个月,膝关节X线片显示股骨外侧con骨破坏和胫骨前骨破坏。胫骨结节缺失。胫骨前部的胫骨结节很少消失。如果发生这种情况,则很难重建并且全膝关节置换术变得复杂。对于胫骨中的骨缺损,使用胶结剂来重建胫骨前表面的形状。有可能最小化骨切除的体积并在形态上重建胫骨结节。病人很快康复了。术后第5周,患者能够用拐杖走路。全膝关节置换术后36个月,膝盖伸展度为0°,屈曲度为120°,伸展滞后为5°,膝关节评分从40分提高到94分,功能评分从20分提高到75分。但是,长期植入物的稳定性需要仔细监测。

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