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Intramyocardial dissecting hematoma and postinfarction cardiac rupture

机译:心肌内夹层血肿和梗塞后心脏破裂

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Potentially fatal cardiac rupture is a complication of myocardial infarction (MI), which can appear in the first hours of the acute event and during the course of the first week. The intramyocardial dissecting hematoma might appear as a component of the rupture during the evolution process. The description of the myocardium as a helical muscular band facilitates the explanation of the fiber dissection. With echocardiography, it is possible to diagnose intramyocardial dissecting hematomas (IDH), determine its location, progression, potential complications, and in some cases its reabsorption. It is necessary to search for neocavitations in the infarcted myocardium and identify the intramyocardial edge that surrounds the defect, as well as the flow inside the myocardial dissection, the pathway of the dissection, and its communication with ventricular cavities, and also to look for the complete or partial reabsorption of the cavitary image. The greater the myocardial dissection is, the worse the prognosis. If the dissecting hematoma is confined to the apical segments, it is more likely to reabsorb spontaneously. Tissue characterization with magnetic resonance during an acute myocardial infarction allows identification of reperfusion injuries with altered microcirculation and intramyocardial hemorrhage (IMH). It is necessary to search for IMH in reperfused patients with ventricular arrhythmias, stunned myocardium, and no reflow. These patients may develop an increased stiffness in the infarcted wall and a major likelihood to develop a parietal rupture. Everything seems to indicate that we are facing the same physiopathological process which can be characterized by 2 complementary imaging methods, echocardiography and magnetic resonance.
机译:潜在的致命性心脏破裂是心肌梗塞(MI)的并发症,可在急性事件的最初几个小时和第一周的过程中出现。心肌内夹层血肿可能在进化过程中表现为破裂的一部分。将心肌描述为螺旋形肌肉带有助于解释纤维解剖。借助超声心动图,可以诊断心肌内夹层血肿(IDH),确定其位置,进展,潜在并发症以及某些情况下的重吸收。有必要在梗塞的心肌中寻找新的空化,并确定围绕缺损的心肌内边缘,以及心肌夹层内的血流,夹层的通路及其与心腔的连通,并寻找完全或部分重吸收空化图像。心肌夹层越大,预后越差。如果解剖性血肿仅限于根尖段,则更有可能自发吸收。急性心肌梗死期间具有磁共振的组织特征可以识别微循环和心肌内出血(IMH)改变的再灌注损伤。对于有心律失常,心肌昏迷且无再流的再灌注患者,必须寻找IMH。这些患者可能会增加梗死壁的僵硬程度,并且很有可能发生壁突破裂。一切似乎都表明我们正面临着相同的生理病理过程,这可以通过两种互补的成像方法,超声心动图和磁共振来表征。

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