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Repeated Hypoxic Syncope in a Helicopter Pilot at a Simulated Altitude of 18,000 Feet

机译:在模拟高度18,000英尺的直升机飞行员中反复缺氧性晕厥

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Background: Military aircrew members undergo regular aviation physiology training, including hypobaric chamber flight to demonstrate the effects of hypoxia at altitude on human physiology and to strengthen hypoxia awareness. Premature near syncope under hypoxia will jeopardize flight safety. However, the causes of such abnormality may not be apparent. Case Report: We present a case of a healthy army helicopter pilot who suffered repeated premature near syncope and bradycardia during chamber flight at 18,000 ft (5486 m). He had no previous record of fainting. A passive head-up tilt table study under normoxia revealed cardiovascular responses compatible with vasovagal syncope. After considering the nature of the abnormality, mission characteristics, and the operating altitude for the helicopter, the pilot was granted a restricted waiver, with missions limited to below 10,000 ft (3048 m) in multicrew helicopters. Discussion: Evaluation of hypoxic syncope focuses on cardiovascular, neurologic, and psychiatric systems. The possibility of newly acquired dysautonomia as the cause of hypoxia syncope was supported by normal hypoxia tolerance in previous chamber flights and the results of the head-up tilt table study. The possible pathophysiology of hypoxic syncope and its association with a vasovagal constitution is presented. For a previously healthy helicopter pilot, impaired hypoxia tolerance may cause in-flight incapacitation. The cause should be investigated and the possibility of recurrence in flight should be evaluated before an adequate aeromedical decision can be made.
机译:背景:军用机组人员接受定期的航空生理训练,包括低压舱飞行,以证明高原缺氧对人体生理的影响并增强缺氧意识。缺氧时过早接近晕厥会危害飞行安全。但是,这种异常的原因可能并不明显。病例报告:我们介绍了一个健康的陆军直升机飞行员的案例,该飞行员在18,000英尺(5486 m)的舱室飞行期间反复遭受晕厥和心动过缓的反复发作。他以前没有昏厥的记录。一项在常氧下进行的被动式平视倾斜表研究显示,心血管反应与血管迷走性晕厥相吻合。在考虑了异常的性质,任务特征和直升机的工作高度之后,飞行员获得了豁免,在多人直升机中的任务限制在10,000英尺(3048 m)以下。讨论:缺氧性晕厥的评估重点在于心血管,神经系统和精神科系统。在先前的舱室飞行中,正常的低氧耐受性以及平视倾斜表研究的结果支持了新近出现的自主神经功能障碍引起的低氧晕厥的可能性。提出了低氧性晕厥的可能病理生理学及其与血管迷走神经组成的关系。对于以前健康的直升机飞行员,耐缺氧能力受损可能会导致机上失能。在做出充分的航空医学决策之前,应调查原因并评估飞行复发的可能性。

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