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机译:PD-L1和IAPS合作以保护来自细胞毒性淋巴细胞衍生TNF的肿瘤
Peter MacCallum Canc Ctr Canc Immunol Div Immune Def Lab Melbourne Vic 3000 Australia;
Walter &
Eliza Hall Inst Med Res Cell Signalling &
Cell Death Div 1G Royal Parade Melbourne Vic;
Peter MacCallum Canc Ctr Canc Immunol Div Immune Def Lab Melbourne Vic 3000 Australia;
Peter MacCallum Canc Ctr Canc Immunol Div Immune Def Lab Melbourne Vic 3000 Australia;
Walter &
Eliza Hall Inst Med Res Cell Signalling &
Cell Death Div 1G Royal Parade Melbourne Vic;
Peter MacCallum Canc Ctr Canc Immunol Div Immune Def Lab Melbourne Vic 3000 Australia;
机译:PD-L1和IAPS合作以保护来自细胞毒性淋巴细胞衍生TNF的肿瘤
机译:IAP拮抗剂和TNF-α溶瘤病毒的组合可诱导肿瘤血管关闭和肿瘤消退
机译:IAP的糖皮质激素依赖性表达参与免受TNF介导的MCF7细胞细胞毒性的保护
机译:HDX / MS分析人肿瘤坏死因子-α(TNFα)和抗TNFα之间的相互作用
机译:肿瘤巨噬细胞的PD-L1表达:调节和信号
机译:PD-L1和IAP协同保护肿瘤免受细胞毒性淋巴细胞衍生坏死因子
机译:c-IAP1和c-IAP2是肿瘤坏死因子α的关键介质 (TNFα)诱导的NF-κB 激活*S⃞
机译:通过肿瘤特异性细胞毒性T细胞分泌工程化肿瘤特异性免疫毒素,增强根除淋巴瘤