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机译:创伤性脑损伤后NLRS和AIM2炎炎γ脑屏障受损血脑屏障的病理作用
Department of Neurosurgery Tianjin Medical University General Hospital;
Laboratory of Neuro-Trauma and Neurodegenerative Disorders Tianjin Geriatrics Institute Tianjin;
Laboratory of Neuro-Trauma and Neurodegenerative Disorders Tianjin Geriatrics Institute Tianjin;
Laboratory of Neuro-Trauma and Neurodegenerative Disorders Tianjin Geriatrics Institute Tianjin;
Department of Neurosurgery Tianjin Medical University General Hospital;
Key Laboratory of Injuries Variations and Regeneration of Nervous System Tianjin Neurological;
Laboratory of Neuro-Trauma and Neurodegenerative Disorders Tianjin Geriatrics Institute Tianjin;
Department of Neurology Duke University Medical Center;
Department of Neurosurgery Tianjin Medical University General Hospital;
Laboratory of Neuro-Trauma and Neurodegenerative Disorders Tianjin Geriatrics Institute Tianjin;
Traumatic brain injury; Blood-brain barrier; Pyroptosis; Inflammasome; NOD-like receptors; AIM2;
机译:创伤性脑损伤后NLRS和AIM2炎炎γ脑屏障受损血脑屏障的病理作用
机译:细胞凋亡在创伤性脑损伤后长期轴突,微血管和血脑屏障损伤中的作用
机译:在创伤性脑损伤后,受伤脑微血管内皮细胞中的miR-21-3p增加通过促进细胞凋亡和通过靶向mat2b来加剧血脑屏障损伤
机译:血液改变的分子成像和血脑屏障中断对脑脑损伤小鼠模型中的影响
机译:休克超压对血脑屏障渗透性在爆炸诱导的创伤性脑损伤中的时间和空间效应
机译:bFGF通过创伤性脑损伤后PI3K-Akt-Rac1途径通过连接蛋白调节来防止血脑屏障损害。
机译:儿茶素衰减创伤性脑损伤诱导的血脑屏障损伤,并改善大鼠的长期神经系统结果
机译:爆炸致颅脑损伤大鼠血脑屏障通透性,氧化应激和活化小胶质细胞增加