机译:1,25(OH)(2)D-3诱导的维生素D受体与NF-Kappa B的P50亚基的相互作用抑制了KLF5和P50之间的相互作用,有助于抑制LPS诱导的巨噬细胞增殖
Hebei Med Univ Minist Educ Key Lab Neural &
Vasc Biol Dept Biochem &
Mol Biol Shijiazhuang;
Hebei Med Univ Minist Educ Key Lab Neural &
Vasc Biol Dept Biochem &
Mol Biol Shijiazhuang;
Hebei Med Univ Hosp 2 Dept Neurol Shijiazhuang 050017 Peoples R China;
Handan First Hosp Dept Pediat Handan 056000 Hebei Peoples R China;
North China Univ Sci &
Technol Sch Publ Hlth Tangshan 063000 Hebei Peoples R China;
North China Univ Sci &
Technol Sch Publ Hlth Tangshan 063000 Hebei Peoples R China;
Hebei Med Univ Minist Educ Key Lab Neural &
Vasc Biol Dept Biochem &
Mol Biol Shijiazhuang;
Hebei Med Univ Minist Educ Key Lab Neural &
Vasc Biol Dept Biochem &
Mol Biol Shijiazhuang;
Hebei Med Univ Minist Educ Key Lab Neural &
Vasc Biol Dept Biochem &
Mol Biol Shijiazhuang;
1; 25(OH)(2)D-3; Vitamin D receptor; NF-kappa B; KLF5; Macrophage; Proliferation;
机译:1,25(OH)(2)D-3诱导的维生素D受体与NF-Kappa B的P50亚基的相互作用抑制了KLF5和P50之间的相互作用,有助于抑制LPS诱导的巨噬细胞增殖
机译:通过在小鼠中删除NF-κB的p50亚基,抑制2,2',4,4',5,5'-六氯联苯(PCB-153)促进肝癌发生。
机译:IL-10对肝炎性结肠炎的抑制作用需要NF-κB的p50 / p105亚基。
机译:通过氢氘交换质谱法研究粒细胞-巨噬细胞集落刺激因子与其受体的可溶性α亚基之间的结合相互作用
机译:突变型p50突变体对NF-κBp50亚基的突变分析和对NF-κB活性的抑制。
机译:NF-Kappa B的P50亚基的突变分析及反式显性P50突变体抑制NF-κB活性的抑制作用