机译:缺氧诱导因子-1α通过在椎间盘变性期间通过Nucleus purposus细胞中的Notch1信号传导介导骨髓和胶原Pi表达
Shanghai Jiao Tong Univ Sch Med Ruijin Hosp Dept Orthoped Shanghai Peoples R China;
Shanghai Jiao Tong Univ Sch Med Ruijin Hosp Dept Orthoped Shanghai Peoples R China;
Shanghai Jiao Tong Univ Sch Med Ruijin Hosp Dept Orthoped Shanghai Peoples R China;
Fifth Peoples Hosp Jinan Dept Orthoped Jinan Peoples R China;
Shanghai Jiao Tong Univ Sch Med Ruijin Hosp Dept Orthoped Shanghai Peoples R China;
Shanghai Jiao Tong Univ Sch Med Ruijin Hosp Dept Orthoped Shanghai Peoples R China;
Shanghai Jiao Tong Univ Sch Med Ruijin Hosp Dept Orthoped Shanghai Peoples R China;
Shanghai Jiao Tong Univ Sch Med Ruijin Hosp Dept Orthoped Shanghai Peoples R China;
Shanghai Jiao Tong Univ Sch Med Ruijin Hosp Dept Orthoped Shanghai Peoples R China;
HIF-1 alpha; Nucleus pulposus cells; Aggrecan; Collagen Pi; NOTCH1;
机译:缺氧诱导因子-1α通过在椎间盘变性期间通过Nucleus purposus细胞中的Notch1信号传导介导骨髓和胶原Pi表达
机译:骨形态发生蛋白2通过介导ECM的降解通过PI3K / AKT途径介导ECM和细胞核髓鞘细胞凋亡的椎间盘变性
机译:LIM矿化蛋白1通过维持髓核细胞外基质产生和抑制基质金属蛋白酶表达抑制TNF-α诱导的椎间盘退变。
机译:髓核组织工程治疗椎间盘退变的研究进展
机译:组织工程环纤维纤维素修复和颈椎间盘预原前临床前模型中的细胞核脉搏增强
机译:PI3K / AKT通过调节椎间盘的细胞核牙髓细胞中的SOX9表达和活性来调节聚集体基因表达
机译:TGFβ通过核心牙髓细胞中的规范Smad3信号通路调节Galectin-3表达:椎间盘变性中的含义