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机译:MEG3通过PI3K / mTOR途径抑制上皮 - 间充质转换来影响T细胞淋巴细胞淋巴瘤的进展和化学抑制
Department of HematologyHematopoietic Stem Cell Transplantation/Cell Immunotherapy Center Cheng Du;
Department of HematologyHematopoietic Stem Cell Transplantation/Cell Immunotherapy Center Cheng Du;
Department of HematologyHematopoietic Stem Cell Transplantation/Cell Immunotherapy Center Cheng Du;
Department of HematologyHematopoietic Stem Cell Transplantation/Cell Immunotherapy Center Cheng Du;
Department of HematologyHematopoietic Stem Cell Transplantation/Cell Immunotherapy Center Cheng Du;
Department of HematologyHematopoietic Stem Cell Transplantation/Cell Immunotherapy Center Cheng Du;
Department of HematologyHematopoietic Stem Cell Transplantation/Cell Immunotherapy Center Cheng Du;
drug resistance; epithelial‐mesenchymal transition; invasion; long noncoding RNA; maternally expressed gene 3; migration; T‐cell lymphoblastic lymphoma;
机译:MEG3通过PI3K / mTOR途径抑制上皮 - 间充质转换来影响T细胞淋巴细胞淋巴瘤的进展和化学抑制
机译:通过反转上皮 - 间充质转换和降低癌症干细胞标志物表达,抑制pi3k / akt / mtor信号传导途径通过逆转上皮 - 间充质转换和降低癌症干细胞标志物表达
机译:TGF-beta(2)通过激活PI3K / Akt / mTOR信号传导途径在培养的人晶状体上皮细胞中诱导上皮-间质转化
机译:PI3K途径在抗IgM(抗IgM(抗-Ig)的作用 - 最终和 - 抗生素B细胞淋巴瘤未脱离PI3K途径信号传导对CH12 B细胞淋巴瘤的I-FI阻力
机译:靶向MEK5,MEK1 / 2和/或PI3K途径将上皮细胞转型的上皮性反转,并通过间充质表型提高乳腺癌和脑癌中的化疗敏感性
机译:雷公藤甲素通过调节PI3K / AKT / mTOR途径抑制细胞活力侵袭和上皮-间质转化从而对T细胞淋巴母细胞瘤具有抗肿瘤作用
机译:通过调节PI3K / AKT / MTOR途径抑制细胞活力,侵袭和上皮间淋巴瘤T细胞淋巴细胞淋巴瘤抗肿瘤效应