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Antitumor effect of triptolide in T-cell lymphoblastic lymphoma by inhibiting cell viability invasion and epithelial–mesenchymal transition via regulating the PI3K/AKT/mTOR pathway

机译：雷公藤甲素通过调节PI3K / AKT / mTOR途径抑制细胞活力侵袭和上皮-间质转化从而对T细胞淋巴母细胞瘤具有抗肿瘤作用

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摘要

IntroductionT-cell lymphoblastic lymphoma (T-LBL) is a widely disseminated disease worldwide. Triptolide (TPL) is purified from Chinese herb and displays anti-inflammatory, anti-fertility, anti-tumor and immunosuppressive effects.

机译：简介T细胞淋巴母细胞淋巴瘤（T-LBL）是世界范围内广泛传播的疾病。雷公藤甲素（TPL）是从中草药中提纯的，具有抗炎，抗生育，抗肿瘤和免疫抑制作用。

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期刊名称 OncoTargets and therapy
作者
Yan Huang; Sun Wu; Yuan Zhang; Lihua Wang; Yan Guo;
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作者单位

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年(卷),期 2018(11),-1
年度 2018
页码 769–779
总页数 11
原文格式 PDF
正文语种
中图分类应用微生物学;肿瘤学;
关键词
triptolide T-cell lymphoblastic lymphoma invasion EMT PI3K/AKT/mTOR;

机译：雷公藤内酯醇;T细胞淋巴母细胞淋巴瘤;侵袭;EMT;PI3K / AKT / mTOR;

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专利

1. Antitumor effect of triptolide in T-cell lymphoblastic lymphoma by inhibiting cell viability, invasion, and epithelial–mesenchymal transition via regulating the PI3K/AKT/mTOR pathway [J] . Yan Huang, Sun Wu, Yuan Zhang, OncoTargets and therapy . 2018,第2期

机译：雷公藤甲素通过调节PI3K / AKT / mTOR途径抑制细胞活力，侵袭和上皮间质转化，从而对T细胞淋巴母细胞瘤具有抗肿瘤作用
2. Scutellarin inhibits the invasive potential of malignant melanoma cells through the suppression epithelial-mesenchymal transition and angiogenesis via the PI3K/Akt/mTOR signaling pathway [J] . Li Chun-Yu, Wang Qi, Wang Xiaomin, European Journal of Pharmacology: An International Journal . 2019,第期

机译：Scutellarin通过PI3K / AKT / MTOR信号通路通过抑制上皮 - 间充质转换和血管生成来抑制恶性黑色素瘤细胞的侵入性潜力
3. MEG3 affects the progression and chemoresistance of T‐cell lymphoblastic lymphoma by suppressing epithelial‐mesenchymal transition via the PI3K/mTOR pathway [J] . Deng Rui, Fan Fang‐Yi, Yi Hai, Journal of cellular biochemistry. . 2019,第5期

机译：MEG3通过PI3K / mTOR途径抑制上皮 - 间充质转换来影响T细胞淋巴细胞淋巴瘤的进展和化学抑制
4. Combination of PI3K/Akt/mTOR inhibitors and PDT in endothelial and tumor cells. [C] . Fateye, Babasola, Chen Bin Optical methods for tumor treatment and detection: mechanisms and techniques in photodynamic therapy XX . 2011

机译：内皮细胞和肿瘤细胞中PI3K / Akt / mTOR抑制剂和PDT的组合。
5. Crosstalk between the TGF-beta/Smad and PI3K/Akt pathways in the human epithelial MCF10A cell lineage and implications of signaling inhibitors. [D] . Zheng, Jie. 2009

机译：人上皮MCF10A细胞谱系中TGF-beta / Smad和PI3K / Akt通路之间的串扰和信号抑制剂的含义。
6. MiR-1224-5p Activates Autophagy Cell Invasion and Inhibits Epithelial-to-Mesenchymal Transition in Osteosarcoma Cells by Directly Targeting PLK1 Through PI3K/AKT/mTOR Signaling Pathway [O] . Bicheng Jin, Dongfang Jin, Zhaozhen Zhuo, 2020

机译：MiR-1224-5P通过直接靶向PLK1通过PI3K / AKT / MTOR信号通路激活自噬细胞侵入并抑制骨肉瘤细胞中的上皮点到间充质转变
7. Antitumor effect of triptolide in T-cell lymphoblastic lymphoma by inhibiting cell viability, invasion, and epithelialndash;mesenchymal transition via regulating the PI3K/AKT/mTOR pathway [O] . Yan Huang, Sun Wu, Yuan Zhang, 2018

机译：通过调节PI3K / AKT / MTOR途径抑制细胞活力，侵袭和上皮间淋巴瘤T细胞淋巴细胞淋巴瘤抗肿瘤效应

Antitumor effect of triptolide in T-cell lymphoblastic lymphoma by inhibiting cell viability invasion and epithelial–mesenchymal transition via regulating the PI3K/AKT/mTOR pathway

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