机译:CD56明亮的天然杀手细胞通过细胞分解和CCCDNA衰减在长期Entecavir治疗的患者切换到Peginterferon Alfa-2a的患者中诱导HBsAg降低
Department and Institute of Infectious DiseaseHuazhong University of Science and TechnologyWuhan;
Department and Institute of Infectious DiseaseHuazhong University of Science and TechnologyWuhan;
Department and Institute of Infectious DiseaseHuazhong University of Science and TechnologyWuhan;
Department and Institute of Infectious DiseaseHuazhong University of Science and TechnologyWuhan;
Department and Institute of Infectious DiseaseHuazhong University of Science and TechnologyWuhan;
Department and Institute of Infectious DiseaseHuazhong University of Science and TechnologyWuhan;
Department of Pediatric DiseaseHuazhong University of Science and TechnologyWuhan China;
Department and Institute of Infectious DiseaseHuazhong University of Science and TechnologyWuhan;
Department and Institute of Infectious DiseaseHuazhong University of Science and TechnologyWuhan;
chronic hepatitis B; covalently closed circular DNA; hepatitis B surface antigen; natural killer cell; peginterferon alfa‐2a;
机译:CD56明亮的天然杀手细胞通过细胞分解和CCCDNA衰减在长期Entecavir治疗的患者切换到Peginterferon Alfa-2a的患者中诱导HBsAg降低
机译:CD56明亮的天然杀手细胞通过细胞分解和CCCDNA衰减在长期Entecavir治疗的患者切换到Peginterferon Alfa-2a的患者中诱导HBsAg降低
机译:PEG-Interferonα诱导CD56(明亮)天然杀手细胞介导的恩替卡韦治疗慢性乙型肝炎患者HBsAg的减少
机译:受体曲目中的明显签名鉴别CD56Bright和CD56Dim天然杀伤细胞
机译:慢性髓性白血病患者CD56 +明亮和CD56 +昏暗的天然杀伤细胞数量逐渐减少,对募集克隆源性杀伤细胞的刺激作出响应,并且表现出每种细胞的增殖降低