机译:褪黑激素的高葡萄糖诱导的粉红色1表达的增强刺激神经元细胞存活:MT 2 2 / AKT / NF-κB途径的参与
Department of Veterinary PhysiologySeoul National UniversitySeoul Korea;
Department of Veterinary PhysiologySeoul National UniversitySeoul Korea;
Department of Veterinary PhysiologySeoul National UniversitySeoul Korea;
Department of Veterinary PhysiologySeoul National UniversitySeoul Korea;
Department of Veterinary PhysiologySeoul National UniversitySeoul Korea;
Department of Veterinary PhysiologySeoul National UniversitySeoul Korea;
Department of Pharmaceutical EngineeringDaegu Haany UniversityGyeongsan South Korea;
Department of Veterinary PhysiologySeoul National UniversitySeoul Korea;
Department of Veterinary PhysiologySeoul National UniversitySeoul Korea;
diabetes mellitus; glucose; melatonin; mitophagy; neuronal cell apoptosis; PTEN‐induced putative kinase 1;
机译:褪黑激素的高葡萄糖诱导的粉红色1表达的增强刺激神经元细胞存活:MT 2 2 / AKT / NF-κB途径的参与
机译:褪黑素可防止细胞凋亡并增强由热激在HL-60细胞中诱导的HSP27 mRNA表达:MT2受体可能参与其中。
机译:PI-3K /Akt-NF-κB信号通路参与缺氧诱导的有丝分裂因子刺激的内皮细胞Flk-1表达
机译:依赖Akt1的途径参与调节肿瘤条件培养基诱导的内皮细胞迁移和存活。
机译:开发可诱导细胞模型系统以研究MT 1和MT2褪黑激素受体。
机译:棕榈酸-BSA通过GPR40介导的神经元细胞双重途径增强淀粉样β的产生:涉及Akt / mTOR /HIF-1α和Akt /NF-κB途径
机译:褪黑素的高葡萄糖诱导的粉红色1表达的增强刺激神经元细胞存活:MT2 / AKT / NF-κB途径的累积