机译:YAR作为致癌蛋白,通过激活PI3K-AKT信号传导来促进胃癌进展
Peking Univ Dept Gastrointestinal Oncol Key Lab Carcinogenesis &
Translat Res Minist Educ;
Peking Univ Dept Gastrointestinal Oncol Key Lab Carcinogenesis &
Translat Res Minist Educ;
Peking Univ Dept Gastrointestinal Oncol Key Lab Carcinogenesis &
Translat Res Minist Educ;
Peking Univ Dept Gastrointestinal Oncol Key Lab Carcinogenesis &
Translat Res Minist Educ;
Peking Univ Dept Gastrointestinal Oncol Key Lab Carcinogenesis &
Translat Res Minist Educ;
Peking Univ Dept Gastrointestinal Oncol Key Lab Carcinogenesis &
Translat Res Minist Educ;
Peking Univ Dept Gastrointestinal Oncol Key Lab Carcinogenesis &
Translat Res Minist Educ;
Peking Univ Dept Gastrointestinal Oncol Key Lab Carcinogenesis &
Translat Res Minist Educ;
Peking Univ Dept Gastrointestinal Oncol Key Lab Carcinogenesis &
Translat Res Minist Educ;
Peking Univ Dept Gastrointestinal Oncol Key Lab Carcinogenesis &
Translat Res Minist Educ;
Gastric cancer; Oncogenic protein; YARS; PI3K-Akt; Homologous recombination;
机译:YAR作为致癌蛋白,通过激活PI3K-AKT信号传导来促进胃癌进展
机译:MicroRNA-423-3P-BIM轴促进癌症进展,并在胃癌中激活致癌自噬
机译:炭疽毒素受体1 /肿瘤内皮标记物8通过激活PI3K / AKT / MTOR信号通路促进胃癌进展
机译:使用生物信息学方法探讨蛋白质表达与亚细定位的连接和胃癌进展
机译:利用蛋白质进化来阐明致癌细胞因子信号轴,并为高度致命癌症开发新的治疗剂
机译:细胞内基质Gla蛋白通过激活JAK2 / STAT5信号传导促进胃癌的肿瘤进展
机译:细胞内基质Gla蛋白通过激活JAK2 / Stat5信号传导在胃癌中促进肿瘤进展