机译:降低ola1(obm-lib-sapase-1)表达驱动泛素 - 蛋白酶体途径,以在新生儿的持续肺动脉高压下下调线粒体SOD2(超氧化物歧化酶)
Med Coll Wisconsin Dept Pediat Div Neonatol Cardiovasc Res Ctr Childrens Res Inst 8701;
Children Hosp Wisconsin Dept Pediat Milwaukee WI USA;
Med Coll Wisconsin Dept Pediat Div Neonatol Cardiovasc Res Ctr Childrens Res Inst 8701;
Children Hosp Wisconsin Dept Pathol Milwaukee WI USA;
Shanghai Jemincare Pharmaceut Co Ltd Shanghai Peoples R China;
Children Hosp Wisconsin Dept Pediat Milwaukee WI USA;
Med Coll Wisconsin Dept Surg Div Pediat Surg 8700 W Wisconsin Ave Milwaukee WI 53226 USA;
Med Coll Wisconsin Dept Pediat Div Neonatol Cardiovasc Res Ctr Childrens Res Inst 8701;
Med Coll Wisconsin Dept Pediat Div Neonatol Cardiovasc Res Ctr Childrens Res Inst 8701;
apoptosis; caspase; hypoxemia; mitochondria; oxidative stress; reactive oxygen species;
机译:降低ola1(obm-lib-sapase-1)表达驱动泛素 - 蛋白酶体途径,以在新生儿的持续肺动脉高压下下调线粒体SOD2(超氧化物歧化酶)
机译:OLA1(Obg样ATPase-1)表达减少驱动泛素-蛋白酶体途径下调新生儿持续性肺动脉高压中的线粒体SOD2(超氧化物歧化酶)
机译:锰超氧化物歧化酶表达和活性的下降有助于新生儿持续性肺动脉高压的氧化应激
机译:锰超氧化物歧化酶表达和活性的下降有助于新生儿持续性肺动脉高压的氧化应激
机译:降低OLA1(ob样ATPase-1)表达驱动泛素 - 蛋白酶体途径,使新生儿持续肺动脉高压下测量线粒体SOD2(超氧化物歧化酶)