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Is Addiction a Heterogeneous Condition? Reflections on Pickard's 'The Purpose in Chronic Addiction'

机译:成瘾是一种异质性疾病吗?对皮卡德的“长期成瘾的目的”的思考

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Both the traditional disease model of addiction and Pickard's "purposive" model (Pickard 2012) attempt to define the motivational influence exerted by substances of abuse on agents that influence the agents' behavior toward further substance consumption. Such a definition would seem to take the form "For all substances S that cause addiction/dependence, there exists a kind of motivational influence M such that agents affected by M are more inclined to engage in substance-related behaviors B." In these terms, the argument between Pickard and proponents of the disease model is over the nature of M, and evidence such as the epidemiological data cited by Pickard and neurological changes cited by disease theories (e.g., Everitt and Robbins 2005) are attempts to support differing conceptions of M. Specifically, the disease model argues that -M is a change in control from mechanisms underlying voluntary to those underlying compulsive action, whereas Pickard argues that M is a combination of five factors that converge around a putative reason or "purpose" for the agent to consume the substance.
机译:传统的成瘾疾病模型和Pickard的“有目的性”模型(Pickard 2012)都试图定义滥用药物对药物产生的动机影响,这些药物影响药物的行为以进一步消耗药物。这样的定义似乎采取的形式是:“对于所有导致成瘾/依赖性的物质S,都有一种动机影响M,使得受M影响的物质更倾向于从事与物质有关的行为B。”用这些术语,皮卡德和疾病模型的支持者之间的争论是关于M的本质的,而诸如皮卡德引用的流行病学数据和疾病理论所引用的神经系统变化(例如Everitt和Robbins 2005)的证据都在试图支持。具体来说,疾病模型认为-M是控制机制的变化,其机制从自愿机制变为强迫行为,而Pickard认为M是五个因素的组合,这些因素围绕推定的原因或“目的”收敛以便代理商消耗该物质。

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