>Patients with type 2 diabetes mellitus (T2DM) are usually with poor immunity and easier to suffer from cancer and m'/>
机译:Coriolus Versicolor含水提取物改善胰岛素抵抗与PI3K / AKT和P38 MAPK信号传导途径,参与糖尿病骨骼肌
Department of EndocrinologyThe Second affiliated Hospital of Harbin Medical UniversityHarbin Heilongjiang Province 150001 China;
Department of EndocrinologyThe Second affiliated Hospital of Harbin Medical UniversityHarbin Heilongjiang Province 150001 China;
Department of EndocrinologyThe Second affiliated Hospital of Harbin Medical UniversityHarbin Heilongjiang Province 150001 China;
Department of EndocrinologyThe Second affiliated Hospital of Harbin Medical UniversityHarbin Heilongjiang Province 150001 China;
Department of EndocrinologyThe Second affiliated Hospital of Harbin Medical UniversityHarbin Heilongjiang Province 150001 China;
Academician WorkstationHarbin Medical UniversityHarbin Heilongjiang Province 150001 China;
Department of PharmacologyCollege of Pharmacy Harbin Medical UniversityHarbin Heilongjiang Province 150001 China;
Department of EndocrinologyThe Second affiliated Hospital of Harbin Medical UniversityHarbin Heilongjiang Province 150001 China;
Chinese medicine; Coriolus versicolor; diabetic mellitus; glucose transporter 4; insulin resistance; skeletal muscle;
机译:Coriolus Versicolor含水提取物改善胰岛素抵抗与PI3K / AKT和P38 MAPK信号传导途径,参与糖尿病骨骼肌
机译:MKP-1的骨骼肌特异性缺失显示了调节肥胖诱导的胰岛素抵抗的P38 MAPK / JNK / AKT信号节点
机译:1-脱氧野rim霉素通过激活db / db小鼠骨骼肌中的胰岛素信号PI3K / AKT途径来减轻胰岛素抵抗。
机译:使用连续Petri网建模PI3K / PDK1 / Akt和MAPK信号通路
机译:FGF和p38 MAPK信号失调是骨骼肌衰老过程中干细胞自我更新丧失的基础。
机译:人脐带衍生的间充质干细胞通过PTE3K / AKT和ERK / MAPKS信号传导途径在DB / DB小鼠的骨骼肌之间的PTEN介导的串扰来改善胰岛素抗性
机译:人脐带衍生的间充质干细胞通过PTE3K / AKT和ERK / MAPKS信号传导途径在DB / DB小鼠的骨骼肌之间的PTEN介导的串扰来改善胰岛素抗性