机译:Bortezomib通过SMURF1 / AKT / MTOR / P70S6K途径在大鼠同种异体移植肾间质纤维化模型中衰减上皮对间充质转换的进展
Nanjing Med Univ Affiliated Hosp 1 Nanjing Jiangsu Peoples R China;
Nanjing Med Univ Affiliated Hosp 1 Nanjing Jiangsu Peoples R China;
Nanjing Med Univ Affiliated Hosp 1 Nanjing Jiangsu Peoples R China;
Nanjing Med Univ Affiliated Hosp 1 Nanjing Jiangsu Peoples R China;
Nanjing Med Univ Affiliated Hosp 1 Nanjing Jiangsu Peoples R China;
Nanjing Med Univ Affiliated Hosp 1 Nanjing Jiangsu Peoples R China;
Nanjing Med Univ Affiliated Hosp 1 Nanjing Jiangsu Peoples R China;
Nanjing Med Univ Affiliated Hosp 1 Nanjing Jiangsu Peoples R China;
Nanjing Med Univ Affiliated Hosp 1 Nanjing Jiangsu Peoples R China;
机译:Bortezomib通过SMURF1 / AKT / MTOR / P70S6K途径在大鼠同种异体移植肾间质纤维化模型中衰减上皮对间充质转换的进展
机译:硼替佐米通过调节TNF-α-Smurf1-Akt-mTOR-P70S6K途径诱导的EMT减轻肾脏移植中的肾间质纤维化
机译:NRF2信号传导通过PI3K / AKT信号通路衰减上皮 - 间充质过渡和肾间质纤维化
机译:尾加压素II及其受体在大鼠肾间质纤维化中的作用
机译:抑制mtor抑制在肾细胞癌中诱导的代偿性存活和增殖途径激活。
机译:硼替佐米通过调节TNF-α-Smurf1-Akt-mTOR-P70S6K途径诱导的EMT减轻肾脏移植中的肾间质纤维化
机译:通过调节TNF-α-SMURF1-AKT-P70S6K途径诱导的EMT,硼德西布衰减肾移植中的肾间质纤维化纤维化