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机译:长度非编码RNA UCA1的下调参与由N-亚硝基胺联合微囊藻酸酯-LR诱导的HET-1A细胞恶性转化
Southeast Univ Sch Publ Hlth Nanjing Jiangsu Peoples R China;
Southeast Univ Sch Publ Hlth Nanjing Jiangsu Peoples R China;
Southeast Univ Sch Publ Hlth Nanjing Jiangsu Peoples R China;
Southeast Univ Sch Publ Hlth Nanjing Jiangsu Peoples R China;
Southeast Univ Sch Publ Hlth Nanjing Jiangsu Peoples R China;
Southeast Univ Sch Publ Hlth Nanjing Jiangsu Peoples R China;
Southeast Univ Sch Publ Hlth Nanjing Jiangsu Peoples R China;
Southeast Univ Sch Publ Hlth Nanjing Jiangsu Peoples R China;
机译:长度非编码RNA UCA1的下调参与由N-亚硝基胺联合微囊藻酸酯-LR诱导的HET-1A细胞恶性转化
机译:上皮 - 间充质转换和癌症干细胞,由长期非编码RNA,HotaT介导的,参与香烟烟雾提取物诱导的细胞恶性转化
机译:MicroRNA-218通过EzH2介导的H3K27三甲基化的表观遗传沉默参与了香烟烟雾提取物诱导的HBE细胞的恶性转化
机译:难熔性陶瓷纤维诱导支气管上皮细胞恶变
机译:受体酪氨酸激酶EphA2的过表达诱导了良性乳腺上皮的恶性转化:暗示增加细胞存活率和改变乳腺癌细胞粘附。
机译:从PVT1外显子9的长非编码RNA在前列腺癌中过表达并在前列腺上皮细胞中诱导恶性转化和去势抵抗。
机译:长度非编码RNA UCA1通过调节miR-624-3P / VEGF-C促进高葡萄糖诱导的人视网膜内皮细胞血管生成