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机译:CREB / BDNF / TRKB路径的低功能规划在产前地塞米松暴露后在雄性后代中介导认知障碍
Department of Pharmacology School of Basic Medical Sciences Wuhan University;
Department of Pharmacology School of Basic Medical Sciences Wuhan University;
Department of Pharmacology School of Basic Medical Sciences Wuhan University;
Department of Pharmacology School of Basic Medical Sciences Wuhan University;
Department of Pharmacology School of Basic Medical Sciences Wuhan University;
Department of Pharmacology School of Basic Medical Sciences Wuhan University;
Hubei Provincial Key Laboratory of Developmentally Originated Disease;
Department of Pharmacology School of Basic Medical Sciences Wuhan University;
Pregnancy dexamethasone exposure; Hippocampus; Cognitive impairment; Glucocorticoid receptor; Brain-derived neurotrophic factor;
机译:CREB / BDNF / TRKB路径的低功能规划在产前地塞米松暴露后在雄性后代中介导认知障碍
机译:PDGFRβ/ FAK路径的低活动编程介导H型血管发育不良,对产前地塞米松暴露后女性后代大鼠骨质疏松症的高敏感性
机译:IGF1 / MAPK / ERK信号通路介导的肾上腺皮质细胞肾上腺皮质细胞增殖的介导的编程改变,在雄性后代大鼠中的产前咖啡因暴露
机译:产前暴露的2,2,4,4-四溴二苯醚(PBDE 47)诱导雄性后代小鼠精子超氧阴离子的产生和过度活化
机译:伊卡甙II通过抑制淀粉样蛋白生成途径减轻慢性脑灌注不足所致的认知障碍:BDNF / TrkB / CREB信号的参与以及大鼠PPARα和PPARγ的上调
机译:RTMS改善与BDNF / TRKB信号通路相关的男性后代大鼠的产前应激诱导的认知缺陷