机译:缺乏CYP1A1基因的新生小鼠更容易受到氧介导的肺损伤的影响,并被出生的β-萘洛伐克给药救出:在早产儿的支气管肺肺不良的影响
Texas Childrens Hosp Baylor Coll Med Dept Pediat Sect Neonatol Houston TX 77030 USA;
Texas Childrens Hosp Baylor Coll Med Dept Pediat Sect Neonatol Houston TX 77030 USA;
Texas Childrens Hosp Baylor Coll Med Dept Pediat Sect Neonatol Houston TX 77030 USA;
Texas Childrens Hosp Baylor Coll Med Dept Pediat Sect Neonatol Houston TX 77030 USA;
Texas Childrens Hosp Baylor Coll Med Dept Pediat Sect Neonatol Houston TX 77030 USA;
Methodist Hosp Phys Org Dept Pathol &
Genom Med Houston TX 77030 USA;
Texas Childrens Hosp Baylor Coll Med Dept Pediat Sect Neonatol Houston TX 77030 USA;
Texas Childrens Hosp Baylor Coll Med Dept Pediat Sect Neonatol Houston TX 77030 USA;
Texas Childrens Hosp Baylor Coll Med Dept Pediat Sect Neonatol Houston TX 77030 USA;
lung; oxidative injury; beta-naphthoflavone; cytochrome P4501A1; newborn; bronchopulmonary dysplasia;
机译:缺乏CYP1A1基因的新生小鼠更容易受到氧介导的肺损伤的影响,并被出生的β-萘洛伐克给药救出:在早产儿的支气管肺肺不良的影响
机译:细胞色素P4501A1(CYP1A1)的高氧介导的转录激活并降低了新生儿小鼠氧介导的肺损伤的敏感性
机译:新生婴儿的肺损伤和支气管扩张发育不良。
机译:缺乏细胞色素P450(CYP)1A2基因的小鼠显示出增加的F2 - 依泊司蛋白,氧化DNA加合物和体内高氧肺损伤水平
机译:缺乏Cyp1a1基因的新生小鼠更易受氧介导的肺损伤并被产后β-萘黄酮给药挽救:对早产儿支气管肺发育不良的影响
机译:增强氧气介导的细胞色素P450(CYP)1A表达的转录激活和增加在体内携带人CYP1A1或小鼠1A2启动子的转基因小鼠中的高氧肺损伤的敏感性增加