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机译:adipocytes影响阉割的前列腺癌细胞,以在肿瘤细胞中改变PD-L1 / NKG2D配体水平的改变NK细胞对NK细胞的细胞毒性作用的抵抗力
Department of Radiation OncologyUniversity of Rochester School of Medicine and DentistryRochester;
Department of Radiation OncologyUniversity of Rochester School of Medicine and DentistryRochester;
Department of Radiation OncologyUniversity of Rochester School of Medicine and DentistryRochester;
DepThe Second Affiliated Hospital of Soochow UniversitySuzhou Jiangsu P.R. China;
DepThe Second Affiliated Hospital of Soochow UniversitySuzhou Jiangsu P.R. China;
Department of Radiation OncologyUniversity of Rochester School of Medicine and DentistryRochester;
Department of Radiation OncologyUniversity of Rochester School of Medicine and DentistryRochester;
Department of Radiation OncologyUniversity of Rochester School of Medicine and DentistryRochester;
Department of Radiation OncologyUniversity of Rochester School of Medicine and DentistryRochester;
adipocytes; castration‐resistant prostate cancer; IL‐6; leptin; NK cell cytotoxicity; PD‐L1;
机译:adipocytes影响阉割的前列腺癌细胞,以在肿瘤细胞中改变PD-L1 / NKG2D配体水平的改变NK细胞对NK细胞的细胞毒性作用的抵抗力
机译:负载垂死的同种异体前列腺癌细胞的树突状细胞生成抗去势抵抗性前列腺癌细胞的有效细胞毒性T淋巴细胞。
机译:循环肿瘤细胞的全基因组拷贝数改变,具有Abiraatorone或抗甲醛抗性转移性阉割前列腺癌的男性
机译:转移结直肠癌(MCRC)的循环肿瘤细胞(CTC)的单细胞基因组分析识别肿瘤异质性和稀有躯体驾驶员改变
机译:细胞外基质诱导肿瘤存活和抗阉割前列腺癌的耐药机制
机译:在去势抵抗性前列腺癌细胞中抑制IL-6JAK / Stat3信号通过改变PD-1 / NKG2D配体水平来增强NK细胞介导的细胞毒性
机译:辐射改变肺癌细胞中的PD-L1 / NKG2D配体水平,并通过IL-6-MEK / ERK信号通路从NK细胞细胞毒性免疫逸出