机译:内源性ET-1通过在缺氧搏动大鼠Atria中激活COX2-L-PGDS-PPARγ信号来促进ANP分泌
Yanbian Univ Sch Med Sci Dept Physiol Gongyuan Rd 977 Yanji 133002 Jilin Peoples R China;
Yanbian Univ Sch Med Sci Dept Physiol Gongyuan Rd 977 Yanji 133002 Jilin Peoples R China;
Yanbian Univ Sch Med Sci Dept Physiol Gongyuan Rd 977 Yanji 133002 Jilin Peoples R China;
Yanbian Univ Sch Med Sci Dept Physiol Gongyuan Rd 977 Yanji 133002 Jilin Peoples R China;
Yanbian Univ Inst Clin Med Yanji 133000 Peoples R China;
Yanbian Univ Sch Med Sci Dept Physiol Gongyuan Rd 977 Yanji 133002 Jilin Peoples R China;
Hypoxia; Endothelin-1; Cyclooxygenase 2; Lipocalin-type prostaglandin D synthase; Atrial natriuretic peptide;
机译:内源性ET-1通过在缺氧搏动大鼠Atria中激活COX2-L-PGDS-PPARγ信号来促进ANP分泌
机译:内源性内皮素-1通过激活孤立的搏动兔心房中的MAPK / ERK和PI3K / Akt信号通路来调节低氧诱导的心钠素的分泌
机译:HIF-1α-L-PGDS-PPARγ调节缺氧诱导的ANP分泌物在敲打大鼠阿里亚
机译:刻痕信号通路有助于促进大鼠卒中后内源性神经干细胞的增殖
机译:15个脱氧前列腺素J2-甘油(一种2-花生四烯酸甘油的假定的环氧合酶-2代谢产物)对白介素2分泌的抑制作用涉及过氧化物酶体增殖物活化受体γ的活化。
机译:NOX4 / SRC通过激活ERK1 / 2和AKT / GATA4信号来调节ANP分泌在击败大鼠缺氧ATRIA时
机译:NOX4 / SRC通过激活ERK1 / 2和AKT / GATA4信号来调节ANP分泌,在击败大鼠缺氧ATRIA时