机译:GSK-3β磷酸化通过PI3-K / Akt在大鼠脑缺血诱导神经发生中的促进
Department of Applied Biochemistry Tokyo University of Pharmacy and Life Sciences;
Department of Applied Biochemistry Tokyo University of Pharmacy and Life Sciences;
Department of Applied Biochemistry Tokyo University of Pharmacy and Life Sciences;
Department of Applied Biochemistry Tokyo University of Pharmacy and Life Sciences;
Department of Applied Biochemistry Tokyo University of Pharmacy and Life Sciences;
Department of Applied Biochemistry Tokyo University of Pharmacy and Life Sciences;
Department of Molecular and Cellular Pharmacology Tokyo University of Pharmacy and Life Sciences;
Department of Applied Biochemistry Tokyo University of Pharmacy and Life Sciences;
Cerebral ischemia; Neurogenesis; GSK-3β; PI3-K/Akt;
机译:GSK-3β磷酸化通过PI3-K / Akt在大鼠脑缺血诱导神经发生中的促进
机译:白藜芦醇通过PI3-K / Akt途径平行调节GSK-3β和CREB来预防CA1神经元抗缺血性损伤
机译:β-CateninsiRNA抑制成年大鼠大脑短暂性中脑动脉闭塞后缺血诱导的纹状体神经发生。
机译:具有PI3K-AKT路径畸变的患者衍生的乳腺癌异种移植物中AKT同种型的磷酸化位点的平行反应监测测定
机译:中度低温对C57 / Bl6小鼠IL-1β和缺血性脑炎症的影响
机译:通过PI3-K / Akt参与GSK-3β磷酸化在大鼠脑缺血性神经发生中的作用。
机译:通过PI3-K / Akt参与GSK-3β磷酸化在大鼠脑缺血性神经发生中的作用。