机译:MiR-204通过缺血再灌注诱导急性肾脏损伤后靶向SP1来调节上皮 - 间充质转换
Shanghai Jiao Tong Univ Dept Nephrol Xin Hua Hosp Sch Med 1665 Kongjiang Rd Shanghai 200092;
Shanghai Jiao Tong Univ Dept Nephrol Xin Hua Hosp Sch Med 1665 Kongjiang Rd Shanghai 200092;
Shanghai Jiao Tong Univ Dept Nephrol Xin Hua Hosp Sch Med 1665 Kongjiang Rd Shanghai 200092;
Shanghai Jiao Tong Univ Dept Nephrol Xin Hua Hosp Sch Med 1665 Kongjiang Rd Shanghai 200092;
Shanghai Jiao Tong Univ Dept Nephrol Xin Hua Hosp Sch Med 1665 Kongjiang Rd Shanghai 200092;
Shanghai Jiao Tong Univ Dept Nephrol Xin Hua Hosp Sch Med 1665 Kongjiang Rd Shanghai 200092;
Shanghai Jiao Tong Univ Dept Nephrol Xin Hua Hosp Sch Med 1665 Kongjiang Rd Shanghai 200092;
Shanghai Jiao Tong Univ Dept Nephrol Xin Hua Hosp Sch Med 1665 Kongjiang Rd Shanghai 200092;
miR-204; epithelial-mesenchymal transition; acute kidney injury; ischemia-reperfusion; SP1; Chronic-On-AKI;
机译:MiR-204通过缺血再灌注诱导急性肾脏损伤后靶向SP1来调节上皮 - 间充质转换
机译:MiR-204下调SIRT1并逆转SIRT1诱导的上皮-间质转化,对神经的抵抗以及胃癌细胞的侵袭
机译:miR-141通过抑制肾小管上皮细胞中HIPK2表达来调节TGF-β1诱导的上皮-间质转化
机译:PLK1通过诱导上皮 - 间充质转换来促进食管鳞状细胞癌细胞的侵袭
机译:细胞与纤连蛋白的相互作用诱导人乳腺上皮细胞的上皮-间质转化。
机译:长度非编码RNA Neat1通过miR-34a / snail1和miR-204 / zeb1途径调节透镜上皮细胞的TGF-β2诱导的上皮间过渡转变
机译:miR-204通过缺血再灌注诱导急性肾脏损伤后靶向SP1来调节上皮 - 间充质转换