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机译:肿瘤衍生的外泌体教育树突细胞通过HSP72 / HSP105-TLR2 / TLR4途径促进肿瘤转移
Zhejiang Univ Sch Med Inst Immunol Hangzhou 310058 Zhejiang Peoples R China;
Zhejiang Univ Sch Med Inst Immunol Hangzhou 310058 Zhejiang Peoples R China;
Zhejiang Univ Sch Med Inst Immunol Hangzhou 310058 Zhejiang Peoples R China;
Zhejiang Univ Sch Med Inst Immunol Hangzhou 310058 Zhejiang Peoples R China;
Zhejiang Univ Sch Med Inst Immunol Hangzhou 310058 Zhejiang Peoples R China;
Zhejiang Canc Hosp Dept Chemotherapy Hangzhou Zhejiang Peoples R China;
Zhejiang Univ Sch Med Affiliated Hosp 2 Dept Resp Med Hangzhou Zhejiang Peoples R China;
Zhejiang Univ Sch Med Inst Immunol Hangzhou 310058 Zhejiang Peoples R China;
Zhejiang Univ Sch Med Inst Immunol Hangzhou 310058 Zhejiang Peoples R China;
Dendritic cells; Exosomes; HSP72; HSP105; MMP9;
机译:肿瘤衍生的外泌体教育树突细胞通过HSP72 / HSP105-TLR2 / TLR4途径促进肿瘤转移
机译:肿瘤来源的外来体的膜相关Hsp72介导小鼠和人类髓样来源的抑制细胞的STAT3依赖性免疫抑制功能
机译:主要肿瘤衍生的外泌体通过在肝癌中通过Smad3调节循环肿瘤细胞的粘附来促进转移
机译:肿瘤衍生的外泌体基癌症早期检测:分子动力学模拟
机译:探索肿瘤来源的外泌体的药物递送潜力。
机译:肿瘤来源的外泌体通过HSP72 / HSP105-TLR2 / TLR4途径教育树突状细胞促进肿瘤转移
机译:肿瘤衍生的外泌体诱导M2巨噬细胞极化,并通过TIM-3促进骨肉瘤细胞的转移