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机译:突触生理学的长期改变,β2烟碱受体的表达和产前尼古丁暴露大鼠海马中的β2烟碱受体和ERK1 / 2信号传导
Department of Pharmacal Sciences Harrison School of Pharmacy Auburn University Auburn AL 36849;
Department of Pharmacal Sciences Harrison School of Pharmacy Auburn University Auburn AL 36849;
Department of Pharmacal Sciences Harrison School of Pharmacy Auburn University Auburn AL 36849;
Department of Pharmacal Sciences Harrison School of Pharmacy Auburn University Auburn AL 36849;
Department of Pharmacal Sciences Harrison School of Pharmacy Auburn University Auburn AL 36849;
Department of Pharmacal Sciences Harrison School of Pharmacy Auburn University Auburn AL 36849;
Department of Pharmacal Sciences Harrison School of Pharmacy Auburn University Auburn AL 36849;
AMPA receptors; Glutamate; Nicotinic receptors; Single channel; Synaptic plasticity;
机译:产前尼古丁暴露大鼠海马突触生理学,β2烟碱受体表达和ERK1 / 2信号的长期变化
机译:大鼠的产前暴露于尼古丁会导致烟碱胆碱能受体的持续改变。
机译:大鼠的产前暴露于尼古丁会导致烟碱胆碱能受体的持续改变。
机译:茶提取物通过抑制尼古丁和雌激素诱导的9-烟碱乙酰胆碱受体上调在人乳腺癌细胞中赋予其抗增殖效果
机译:大鼠的产前暴露于尼古丁会导致烟碱胆碱能受体的持续改变。
机译:大鼠产前暴露于尼古丁会导致烟碱胆碱能受体持续改变
机译:大鼠对尼古丁的产前暴露导致烟碱胆碱能受体的持续改变
机译:产前暴露于尼古丁和儿童哮喘:尼古丁乙酰胆碱受体,神经肽和纤维连接蛋白在肺中的表达