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机译:Degenic CX26(GJB2)和CX30(GJB6)突变的耳聋机制:通过耳蜗侧壁的异质间隙结函数损伤来降低EndoChlear潜力
Department of Otolaryngology University of Kentucky Medical Center;
Department of Otolaryngology University of Kentucky Medical Center;
Department of Otolaryngology University of Kentucky Medical Center;
Department of Otolaryngology University of Kentucky Medical Center;
Department of Otolaryngology University of Kentucky Medical Center;
Department of Otolaryngology University of Kentucky Medical Center;
Department of Otolaryngology University of Kentucky Medical Center;
Gap junction; Connexin; Cx26; Cx30; Endocochlear potential; Deafness; Cochlea;
机译:Degenic CX26(GJB2)和CX30(GJB6)突变的耳聋机制:通过耳蜗侧壁的异质间隙结函数损伤来降低EndoChlear潜力
机译:间隙连接蛋白Cx26和Cx31突变引起的非综合征性耳聋的双基因遗传。
机译:间隙连接蛋白Cx26和Cx31突变引起的非综合征性耳聋的双基因遗传
机译:双基因Cx26(GJB2)和Cx30(GJB6)突变的耳聋机制:通过损害耳蜗侧壁异质间隙连接功能来降低耳蜗内电位
机译:由Connexin 26(GJB2)缺乏诱导的耳聋不是通过EndoChlear潜力(EP)减少来确定但与耳蜗发育障碍有关