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机译:miR-10a抑制IBD中的树突细胞活化和Th1 / Th17细胞免疫应答
Tongji Univ Shanghai Peoples Hosp 10 Dept Gastroenterol Shanghai 200072 Peoples R China;
Tongji Univ Shanghai Peoples Hosp 10 Dept Gastroenterol Shanghai 200072 Peoples R China;
Tongji Univ Shanghai Peoples Hosp 10 Dept Gastroenterol Shanghai 200072 Peoples R China;
Univ Texas Med Branch Dept Microbiol &
Immunol Galveston TX 77555 USA;
Univ Texas Med Branch Dept Microbiol &
Immunol Galveston TX 77555 USA;
Univ Texas Med Branch Dept Microbiol &
Immunol Galveston TX 77555 USA;
Tongji Univ Shanghai Peoples Hosp 10 Dept Gastroenterol Shanghai 200072 Peoples R China;
Tongji Univ Shanghai Peoples Hosp 10 Dept Gastroenterol Shanghai 200072 Peoples R China;
Univ Texas Med Branch Dept Microbiol &
Immunol Galveston TX 77555 USA;
Univ Texas Med Branch Dept Med Galveston TX 77555 USA;
Univ Texas Med Branch Dept Med Galveston TX 77555 USA;
Tongji Univ Shanghai Peoples Hosp 10 Dept Gastroenterol Shanghai 200072 Peoples R China;
Univ Texas Med Branch Dept Microbiol &
Immunol Galveston TX 77555 USA;
机译:miR-10a抑制IBD中的树突状细胞激活和Th1 / Th17细胞免疫反应
机译:甲酰基肽受体的气道活化通过抑制介体从免疫和炎性细胞释放以及树突状细胞的成熟来抑制Th1和Th17细胞反应
机译:结核分枝杆菌6-kDa蛋白的早期分泌的抗原靶标引发树突状细胞刺激Th17和抑制Th1免疫反应。
机译:Cdsalpha〜+树突状细胞的增强含有肿瘤萃取物的Cdsalpha〜+树突细胞的免疫应答富含热冲击蛋白在肿瘤轴承小鼠中
机译:选择性的CB2受体激活通过减少Th17细胞分化和免疫细胞积累来改善中枢神经系统的炎症。
机译:早期分泌的分枝杆菌抗原蛋白的抗原靶标的分枝杆菌引起的树突细胞刺激TH17并抑制TH1免疫应答
机译:甲醛肽受体的气道激活通过免疫和炎性细胞的抑制介质释放和树突细胞的成熟抑制Th1和Th17细胞反应