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机译:雄激素受体调节的循环循环活性激活KRAS信号以促进膀胱癌侵袭
Cent South Univ Xiangya Hosp Dept Urol Changsha Peoples R China;
Univ Rochester Med Ctr George Whipple Lab Canc Res Dept Pathol Rochester NY 14642 USA;
Cent South Univ Xiangya Hosp Dept Urol Changsha Peoples R China;
Univ Rochester Med Ctr George Whipple Lab Canc Res Dept Pathol Rochester NY 14642 USA;
China Med Univ Hosp Sex Hormone Res Ctr Taichung Taiwan;
Univ Rochester Med Ctr George Whipple Lab Canc Res Dept Pathol Rochester NY 14642 USA;
Univ Rochester Med Ctr George Whipple Lab Canc Res Dept Pathol Rochester NY 14642 USA;
Univ Rochester Med Ctr Dept Orthoped Rochester NY 14642 USA;
Cent South Univ Xiangya Hosp Dept Urol Changsha Peoples R China;
Univ Rochester Med Ctr George Whipple Lab Canc Res Dept Pathol Rochester NY 14642 USA;
bladder cancer; chemo-sensitivity; circular RNA;
机译:雄激素受体调节的循环循环活性激活KRAS信号以促进膀胱癌侵袭
机译:靶向雄激素受体(AR)具有抗抗原醛酰胺酰胺,增加前列腺癌细胞侵袭且通过差异地改变AR / CircRNA-ARC1 / miR-125B-2-3P或MIR-4736 / PPAR伽马/ MMP-9信号来降低膀胱癌细胞侵袭
机译:Wnt7a激活经典Wnt信号,促进膀胱癌细胞入侵,并被miR-370-3p抑制
机译:前列腺癌中雄激素受体的激活 - 蛋白激酶A和细胞外信号调节激酶的作用
机译:核因子-κB通过与雄激素受体信号传导协同促进尿路上皮肿瘤发生和癌症进展。
机译:雄激素受体调节的昼夜循环激活KRAS信号传导以促进膀胱癌入侵
机译:靶向雄激素受体(AR)与抗抗原醛酰胺酰胺增加,通过差异地改变AR / CircRNA-Arc1 / miR-125B-2-3P或MIR-4736 /PPARγ/ MMP-9信号,降低前列腺癌细胞侵袭且膀胱癌细胞侵袭降低