机译:砷促进COX2 / PGE2-SOX2 COX2 / PGE2-SOX2轴以增加尿液细胞的恶性肿瘤性能
Department of Otolaryngology‐Head and Neck SurgeryJohns Hopkins University School of;
The Sidney Kimmel Comprehensive Cancer Johns Hopkins UniversityBaltimore MD;
Department of OncologyJohns Hopkins University School of MedicineBaltimore MD;
Department of PathologyJohns Hopkins University School of MedicineBaltimore MD;
Allegheny Health Network Cancer InstitutePittsburgh PA;
Department of UrologyJohns Hopkins University School of MedicineBaltimore MD;
Department of Otolaryngology‐Head and Neck SurgeryJohns Hopkins University School of;
urothelial carcinoma of bladder; arsenic; cancer stem cell; SOX2; COX2;
机译:砷促进COX2 / PGE2-SOX2 COX2 / PGE2-SOX2轴以增加尿液细胞的恶性肿瘤性能
机译:芳基烃受体的调节通过压制COX2 / PGE2 / Stat3轴来抑制食道鳞状细胞癌进展
机译:芳基烃受体的调节通过压制COX2 / PGE2 / Stat3轴来抑制食道鳞状细胞癌进展
机译:非接触皮秒脉冲电场向上调节间充质干细胞中的SOX2基因表达。
机译:使用QQ改性的SOX2和Neurod1蛋白从人成纤维细胞产生神经干细胞(NSCs)
机译:砷促进COX2 / PGE2-SOX2轴增加尿道上皮细胞的恶性茎特性
机译:免疫效应细胞对COX2 / PGE2轴的激活促进了癌症组织中1型免疫的自限性