机译:去甲基共甾运动(ZST ZST 93)抑制细胞生长,并通过凋亡和自噬途径增强了人类胰腺癌细胞中的吉西他滨的细胞化学敏感性
Department of General SurgeryPeking University First HospitalBeijing People's Republic of China;
Department of General SurgeryPeking University First HospitalBeijing People's Republic of China;
Department of General SurgeryPeking University First HospitalBeijing People's Republic of China;
Department of General SurgeryPeking University First HospitalBeijing People's Republic of China;
Department of General SurgeryPeking University First HospitalBeijing People's Republic of China;
Department of Pharmaceutical ChemistryZhongshan Hospital Fudan UniversityShanghai People's;
Department of Pharmaceutical ChemistryZhongshan Hospital Fudan UniversityShanghai People's;
Department of General SurgeryPeking University First HospitalBeijing People's Republic of China;
ZST93; chemosensitivity; apoptosis; autophagy; pancreatic cancer;
机译:去甲基共甾运动(ZST ZST 93)抑制细胞生长,并通过凋亡和自噬途径增强了人类胰腺癌细胞中的吉西他滨的细胞化学敏感性
机译:miR-17-5p抑制剂通过上调胰腺癌细胞中的Bim表达来增强对吉西他滨的化学敏感性。
机译:通过靶向人胰腺癌细胞中蛋白激酶CK2催化亚基的RNA干扰增强对吉西他滨的化学敏感性
机译:人胰腺癌细胞来源的吉西他滨耐药细胞基因表达分析
机译:OSW-1的抗癌活性:通过钙介导的机制诱导白血病中的凋亡途径和胰腺癌的自噬死亡。
机译:Costunolide通过激活细胞凋亡和自噬途径产生活性氧(ROS)切割Caspase-3和切割Caspase-9来抑制OAW42-A多药耐药人类卵巢癌细胞的生长。
机译:去甲基共沸(ZST93)抑制细胞生长,并通过凋亡和自噬途径增强对人胰腺癌细胞中的吉西他滨的细胞化学敏感性