机译:Fisetin和5-氟尿嘧啶:PIK3CA PIK3CA-矫正结直肠癌的有效组合
Department of DermatologyUniversity of Wisconsin‐MadisonMadison WI;
Department of DermatologyUniversity of Wisconsin‐MadisonMadison WI;
Division of Gastroenterology and Hepatology Department of MedicineUniversity of Wisconsin;
Division of Gastroenterology and Hepatology Department of MedicineUniversity of Wisconsin;
Division of Gastroenterology and Hepatology Department of MedicineUniversity of Wisconsin;
Division of Gastroenterology and Hepatology Department of MedicineUniversity of Wisconsin;
Department of DermatologyUniversity of Wisconsin‐MadisonMadison WI;
Division of Gastroenterology and Hepatology Department of MedicineUniversity of Wisconsin;
Department of Oncology McArdle Laboratory for Cancer ResearchUniversity of Wisconsin;
Department of DermatologyUniversity of Wisconsin‐MadisonMadison WI;
University of Wisconsin Carbone Cancer CenterMadison WI;
colorectal cancer; PIK3CA; 5‐fluorouracil; PI3K/AKT/mTOR signaling;
机译:Fisetin和5-氟尿嘧啶:PIK3CA PIK3CA-矫正结直肠癌的有效组合
机译:TNF-α和氧化石墨烯BEZ235的组合可增强PIK3CA突变型结直肠癌细胞的凋亡
机译:PIK3CA作为乳腺癌癌基因的功能表征综述:1.与乳腺癌相关的PIK3CA突变在乳腺上皮细胞中是致癌的。2.突变型p110α和p110β磷脂酰肌醇3激酶在人乳腺上皮细胞中的致癌特性。
机译:Pik3CA途径和CTNNB1确定右侧转移性结肠直肠癌中西妥昔单抗的良差
机译:突变KRAS和PIK3CA在人类乳腺癌肿瘤发生中的作用。
机译:PI3K / MEK联合抑制可促进PIK3CA野生型KRAS突变性结直肠癌的肿瘤凋亡和消退
机译:Fisetin和5-氟尿嘧啶:PIK3CA-矫正结直肠癌的有效组合