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A likely increase in fine particulate matter and premature mortality under future climate change

机译:在未来的气候变化下,可能会增加细颗粒物质和过早死亡率

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Climate change modulates the concentration of fine particulate matter (PM2.5) via modifying atmospheric circulation, temperature, and the hydrological cycle. Furthermore, PM2.5 is associated with cardiopulmonary diseases and premature mortality. Here, we use seven models to assess the response of PM2.5 to end of the twenty-first century climate change under Representative Concentration Pathway 8.5, and the corresponding impact on premature mortality. The majority of models yield an increase in both PM2.5 and premature mortality associated with lung cancer and cardiopulmonary disease in all world regions except Africa. These results are robust across five different future population projections, although the magnitude of premature deaths can vary by up to a factor of two. Much larger uncertainty is related to uncertainty in model physics and the representation of aerosol processes. Although our analysis requires several assumptions related to future population estimates, as well as the concentration-response function, results suggest that future emission reductions are necessary to avoid the likely health risks associated with increasing PM2.5 in a warmer world.
机译:气候变化通过改变大气循环,温度和水文循环来调节细颗粒物质(PM2.5)的浓度。此外,PM2.5与心肺疾病和过早死亡有关。在这里,我们使用七种模型来评估PM2.5的响应至二十一世纪气候变化的结束,在代表性浓度途径8.5下,对早过死亡率的影响相应影响。大多数模型在除非除非外,大多数模型会产生PM2.5和与肺癌和心肺疾病相关的过早死亡率。这些结果跨越五种不同的未来种群预测,尽管过早死亡的程度可能会变化到两倍。更大的不确定性与模型物理学的不确定性以及气溶胶过程的表示有关。虽然我们的分析需要与未来人口估计有关的几个假设,以及集中响应函数,结果表明,未来的排放减少是必要的,以避免与较温暖的世界中增加PM2.5相关的卫生风险。

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