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机译:ULK1的丧失增加了NR1H / LXR介导的SCD1转录的RPS6KB1-Ncor1抑制,并增加了肝细胞中的脂毒性
Duke NUS Grad Med Sch Cardiovasc &
Metab Disorders Program Lab Hormonal Regulat 8 Coll Rd;
Duke NUS Grad Med Sch Cardiovasc &
Metab Disorders Program Lab Hormonal Regulat 8 Coll Rd;
Duke NUS Grad Med Sch Cardiovasc &
Metab Disorders Program Lab Hormonal Regulat 8 Coll Rd;
Duke NUS Grad Med Sch Cardiovasc &
Metab Disorders Program Lab Hormonal Regulat 8 Coll Rd;
Duke NUS Grad Med Sch Cardiovasc &
Metab Disorders Program Lab Hormonal Regulat 8 Coll Rd;
Duke NUS Grad Med Sch Cardiovasc &
Metab Disorders Program Lab Hormonal Regulat 8 Coll Rd;
Duke NUS Grad Med Sch Cardiovasc &
Metab Disorders Program Lab Hormonal Regulat 8 Coll Rd;
Natl Neurosci Inst Singapore Stroke Trial Unit Singapore Singapore;
Duke NUS Grad Med Sch Cardiovasc &
Metab Disorders Program Lab Hormonal Regulat 8 Coll Rd;
Beth Israel Deaconess Med Ctr Div Endocrinol Diabet &
Metab Boston MA 02215 USA;
Duke NUS Grad Med Sch Cardiovasc &
Metab Disorders Program Lab Hormonal Regulat 8 Coll Rd;
autophagy; lipid droplets; lipotoxicity; LXR; NASH; NCOR1; RPS6KB1; SCD1; ULK1;
机译:ULK1的丧失增加了NR1H / LXR介导的SCD1转录的RPS6KB1-Ncor1抑制,并增加了肝细胞中的脂毒性
机译:ULK1的丧失衰减哺乳动物肝细胞中的胆固醇基因表达
机译:他汀类药物减少了肝癌的表达和分泌,可能通过干扰LXR介导的转录
机译:棕榈酸脂磷脂组合物的改变促进肝脂毒性的ER应激和细胞功能障碍
机译:adipocyte bcl6的转录抑制丧失导致肥胖和胰岛素敏感性
机译:ULK1的丢失会增加RPS6KB1-NCOR1对NR1H / LXR介导的Scd1转录的抑制并增加肝细胞的脂毒性
机译:ULK1的丧失增加了NR1H / LXR介导的SCD1转录的RPS6KB1-Ncor1抑制,并增加了肝细胞中的脂毒性