机译:αB-结晶/ Hspb5通过增强NF-κB诱导的粘附分子ICAM-1,VCAM-1和E-SELETIN的诱导NF-κB诱导的上调诱导内皮 - 白细胞相互作用
Rudbeck Laboratory Department of Immunology Genetics and Pathology Uppsala University 751 85;
Rudbeck Laboratory Department of Immunology Genetics and Pathology Uppsala University 751 85;
Department of Medical Cell Biology Uppsala University 751 23 Uppsala Sweden;
Institute of Anatomy and Cell Biology University of Ulm Albert-Einstein-Allee 11 89081 Ulm;
Department of Medical Cell Biology Uppsala University 751 23 Uppsala Sweden;
Rudbeck Laboratory Department of Immunology Genetics and Pathology Uppsala University 751 85;
αB-crystallin; Chaperone; E-selectin; ICAM-1; NF-κB; VCAM-1;
机译:αb-crystallin/ HspB5通过增强NF-κB诱导的粘附分子ICAM-1,VCAM-1和E-选择素的上调来调节内皮-白细胞相互作用
机译:αB-结晶/ Hspb5通过增强NF-κB诱导的粘附分子ICAM-1,VCAM-1和E-SELETIN的诱导NF-κB诱导的上调诱导内皮 - 白细胞相互作用
机译:αB-crystallin/ HspB5通过增强NF-κB诱导的粘附分子ICAM-1,VCAM-1和E-选择素的上调来调节内皮-白细胞相互作用
机译:靶向淋巴细胞功能相关抗原1(LFA-1)和细胞间粘附分子1(ICAM-1)相互作用,以防止HIV-1的性传播
机译:αB-crystallin/ HspB5通过增强NF-κB诱导的粘附分子ICAM-1VCAM-1和E-选择素的上调来调节内皮-白细胞相互作用
机译:αB-晶状体蛋白/ HspB5通过增强NF-κB诱导的粘附分子ICAM-1,VCAM-1和E-选择素的上调来调节内皮-白细胞相互作用