机译:RH-IFN-alpha通过在实验GMH大鼠模型中通过JAK1-STAT1 / TRAF3途径抑制NF-Kappa B,通过JAK1-Stat1 / Traf3途径来衰减神经炎性并改善神经功能功能
Loma Linda Univ Dept Physiol &
Pharmacol Basic Sci Sch Med Loma Linda CA 92354 USA;
Loma Linda Univ Dept Physiol &
Pharmacol Basic Sci Sch Med Loma Linda CA 92354 USA;
Loma Linda Univ Dept Physiol &
Pharmacol Basic Sci Sch Med Loma Linda CA 92354 USA;
Loma Linda Univ Dept Physiol &
Pharmacol Basic Sci Sch Med Loma Linda CA 92354 USA;
Loma Linda Univ Dept Physiol &
Pharmacol Basic Sci Sch Med Loma Linda CA 92354 USA;
Loma Linda Univ Dept Physiol &
Pharmacol Basic Sci Sch Med Loma Linda CA 92354 USA;
Loma Linda Univ Dept Physiol &
Pharmacol Basic Sci Sch Med Loma Linda CA 92354 USA;
Loma Linda Univ Dept Physiol &
Pharmacol Basic Sci Sch Med Loma Linda CA 92354 USA;
Loma Linda Univ Dept Physiol &
Pharmacol Basic Sci Sch Med Loma Linda CA 92354 USA;
Loma Linda Univ Dept Physiol &
Pharmacol Basic Sci Sch Med Loma Linda CA 92354 USA;
Loma Linda Univ Dept Physiol &
Pharmacol Basic Sci Sch Med Loma Linda CA 92354 USA;
Interferon-alpha; Inflammation; Germinal matrix hemorrhage; Hydrocephalus; Microglia;
机译:RH-IFN-alpha通过在实验GMH大鼠模型中通过JAK1-STAT1 / TRAF3途径抑制NF-Kappa B,通过JAK1-Stat1 / Traf3途径来衰减神经炎性并改善神经功能功能
机译:大麻素受体-2刺激通过在实验GMH大鼠模型中通过CAMP / PKA途径调节微胶质M1 / M2偏振来抑制神经引起的炎症
机译:icariside II通过抑制大鼠的TLR4 / MyD88 / NF-Kappa途径抑制脂多糖诱导的神经炎炎症
机译:一种新型Ultrapotent Poly(ADP-核糖)聚合酶抑制剂改善了慢性心力衰竭大鼠模型中的心脏和内皮功能障碍
机译:使用多尺度实验测试和数值模拟研究在深部地质封存点可能泄漏的过程中浅层含水层中多相CO2释放和衰减的基础研究。
机译:抑制TGF-β可减轻GMH大鼠模型的脑损伤和神经功能缺损
机译:RH-IFN-α通过在实验GMH大鼠模型中抑制NF-κB通过JAK1-STAT1 / TRAF3途径来抑制神经炎炎症并改善神经功能。