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机译:间隙结细胞间通信调节NK细胞活化并调节NK细胞毒性容量
Faculty of Medicine Institute of Biomedical Sciences University of Chile 8380453 Santiago Chile;
Faculty of Medicine Institute of Biomedical Sciences University of Chile 8380453 Santiago Chile;
Millennium Institute on Immunology and Immunotherapy University of Chile 8380453 Santiago Chile;
Faculty of Medicine Institute of Biomedical Sciences University of Chile 8380453 Santiago Chile;
Faculty of Medicine Institute of Biomedical Sciences University of Chile 8380453 Santiago Chile;
Department of Oncology and Pathology Karolinska Institutet 171 76 Stockholm Sweden;
Faculty of Biological Sciences Millennium Institute on Immunology and Immunotherapy Pontifical;
Faculty of Medicine Institute of Biomedical Sciences University of Chile 8380453 Santiago Chile;
Faculty of Biological Sciences Millennium Institute on Immunology and Immunotherapy Pontifical;
Department of Oncology and Pathology Karolinska Institutet 171 76 Stockholm Sweden;
Faculty of Medicine Institute of Biomedical Sciences University of Chile 8380453 Santiago Chile;
Department of Medicine Center for Infectious Medicine Huddinge University Hospital 171 76;
Faculty of Medicine Institute of Biomedical Sciences University of Chile 8380453 Santiago Chile;
机译:间隙结细胞间通信调节NK细胞活化并调节NK细胞毒性容量
机译:间隙连接细胞间通讯调节NK细胞活化并调节NK细胞毒性。
机译:IL-22下调CX43表达,并通过激活牛皮癣的JNK途径降低间隙结肠间通信
机译:HIT-T15细胞胰岛素分泌的增加:透明质酸增强的间隙连接细胞间通信
机译:丝裂素活化的蛋白激酶诱导的心脏间隙连接的磷酸化和缺血后细胞间通讯的调节。
机译:通过调节星形胶质细胞运输连接蛋白43与皮质下囊肿疾病相关的MLC1蛋白的大脑白质脑病有利于间隙连接细胞间的通讯。
机译:间隙连接细胞间通讯调节NK细胞活化并调节NK细胞毒性能力