机译:维生素D在人粘膜组3先天淋巴细胞中下调IL-23受体途径
Center for Infectious Medicine Department of Medicine Huddinge Karolinska Institutet and;
Science for Life Laboratory Department of Medicine;
Center for Infectious Medicine Department of Medicine Huddinge Karolinska Institutet and;
Center for Infectious Medicine Department of Medicine Huddinge Karolinska Institutet and;
Center for Infectious Medicine Department of Medicine Huddinge Karolinska Institutet and;
Science for Life Laboratory Department of Medicine;
Center for Digestive Diseases Department of Medicine Solna Karolinska Institutet and Karolinska;
Department of Molecular Medicine and Surgery;
Department of Otorhinolaryngology Department of Clinical Science Intervention and Technology;
Unit for Inflammation Gastroenterology and Rheumatology Department of Medicine Huddinge;
Center for Infectious Medicine Department of Medicine Huddinge Karolinska Institutet and;
Center for Infectious Medicine Department of Medicine Huddinge Karolinska Institutet and;
Innate lymphoid cells; inflammatory bowel diseases; vitamin D receptor; IL-22;
机译:维生素D在人粘膜组3先天淋巴细胞中下调IL-23受体途径
机译:淋巴毒素控制粘膜病原体攻击过程中肠固有淋巴样细胞的IL-22保护途径。
机译:ILC3-ILC1样细胞的子集在人粘膜组织中产生分集的先天淋巴细胞(VOL 20,PG 980,2019)
机译:胸腺素β_4上调人肝星状细胞中肝细胞生长因子的表达并下调PDGF-β受体的表达
机译:晚期糖基化终产物的受体通过第2组先天淋巴样细胞介导过敏性气道炎症。
机译:CD56的表达标志着人类2组先天性淋巴样细胞与共享的NK细胞和3组先天性淋巴样细胞发育途径的差异
机译:淋巴毒素控制粘膜病原体攻击过程中肠固有淋巴样细胞的IL-22保护途径。