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机译:Rad18独立于体内的FANCONI贫血途径赋予造血祖细胞DNA损伤耐受性
Univ N Carolina Dept Pathol &
Lab Med Chapel Hill NC 27599 USA;
Duke Univ Dept Med Div Hematol Malignancies &
Cellular Therapy Durham NC 27710 USA;
Duke Univ Dept Med Div Hematol Malignancies &
Cellular Therapy Durham NC 27710 USA;
Univ N Carolina Lineberger Comprehens Canc Ctr Carolina Ctr Genome Sci Dept Genet Chapel Hill NC 27599 USA;
Univ N Carolina Dept Pathol &
Lab Med Chapel Hill NC 27599 USA;
Univ N Carolina Lineberger Comprehens Canc Ctr Carolina Ctr Genome Sci Dept Genet Chapel Hill NC 27599 USA;
Duke Univ Dept Biostat &
Bioinformat Durham NC 27710 USA;
Univ N Carolina Dept Pathol &
Lab Med Chapel Hill NC 27599 USA;
Kumamoto Univ IRDA Kumamoto 8600811 Japan;
Univ N Carolina Dept Pathol &
Lab Med Chapel Hill NC 27599 USA;
Kumamoto Univ IMEG Div Cell Maintenance Kumamoto 8600811 Japan;
Duke Univ Dept Med Div Hematol Malignancies &
Cellular Therapy Durham NC 27710 USA;
Univ N Carolina Dept Pathol &
Lab Med Chapel Hill NC 27599 USA;
机译:Rad18独立于体内的FANCONI贫血途径赋予造血祖细胞DNA损伤耐受性
机译:Rad18在体内独立于Fanconi贫血途径赋予造血祖细胞DNA损伤耐受性
机译:Rad18在体内独立于Fanconi贫血途径赋予造血祖细胞DNA损伤耐受性
机译:茎线II造血干细胞膨胀介质中CD34〜+细胞的exvivo扩增产生了大量的功能性早期和晚期祖细胞
机译:范可尼贫血途径在易错DNA损伤耐受性中的作用
机译:Rad18在体内独立于Fanconi贫血途径赋予造血祖细胞DNA损伤耐受性
机译:范可尼贫血中的骨髓衰竭是由加剧造血干细胞和祖细胞的p53 / p21 DNA损伤反应触发的。