to the editor: Complex motor abnormalities in disorders of basal ganglia, such as parkinsonism, often coexist with deficits of eyelid movements. Modest physiology and accessibility have enticed several investigations of eyelid movements in Parkinson's disease. These endeavors are not only useful to characterize phenomenology of degenerative disorders of basal ganglia, but they also have paramount importance in objectively characterizing the course of the disease and understanding the physiological basis of its treatment. Three types of eyelid movement abnormalities are notable in the experimental models of Parkinson's disease: blink hyperrefiexia, impaired blink reflex plasticity, and reduced rate and impaired rhythm of the spontaneous blinks (Basso et al. 1993; Kaminer et al. 2011, 2014, 2015).
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