Donor Hepatic Steatosis Induce Exacerbated Ischemia-Reperfusion Injury Through Activation of Innate Immune Response Molecular Pathways

Gehrau Ricardo C.; Mas Valeria R.; Dumur Catherine I; Suh Jihee L.; Sharma Ashish K.; Cathro Helen P.; Maluf Daniel G.

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Donor Hepatic Steatosis Induce Exacerbated Ischemia-Reperfusion Injury Through Activation of Innate Immune Response Molecular Pathways

机译：供体肝脂肪变性通过激活先天性免疫反应分子途径诱导加重缺血再灌注损伤。

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Background: Severe liver steatosis is a known risk factor for increased ischemia-reperfusion injury (IRI) and poor outcomes after liver transplantation (LT). This study aimed to identify steatosis-related molecular mechanisms associated with IRI exacerbation after LT.

机译：背景：严重的肝脂肪变性是导致缺血再灌注损伤（IRI）增加和肝移植（LT）结果差的已知危险因素。这项研究旨在确定与LT后IRI恶化相关的脂肪变性相关分子机制。

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《Transplantation: Official Journal of the Transplantation Society》 |2015年第12期|共9页
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Gehrau Ricardo C.; Mas Valeria R.; Dumur Catherine I; Suh Jihee L.; Sharma Ashish K.; Cathro Helen P.; Maluf Daniel G.;
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1. Donor Hepatic Steatosis Induce Exacerbated Ischemia-Reperfusion Injury Through Activation of Innate Immune Response Molecular Pathways [J] . Gehrau Ricardo C., Mas Valeria R., Dumur Catherine I, Transplantation: Official Journal of the Transplantation Society . 2015,第12期

机译：供体肝脂肪变性通过激活先天性免疫反应分子途径诱导加重缺血再灌注损伤。
2. Allopurinol induces innate immune responses through mitogen-activated protein kinase signaling pathways in HL-60 cells [J] . Nakajima Akira, Oda Shingo, Yokoi Tsuyoshi Journal of applied toxicology . 2016,第9a10期

机译：Allopurinol通过HL-60细胞中的丝裂剂活化的蛋白激酶信号传导途径诱导先天免疫应答
3. SARS coronavirus spike protein-induced innate immune response occurs via activation of the NF-kappaB pathway in human monocyte macrophages in vitro. [J] . Dosch SF, Mahajan SD, Collins AR Virus Research: An International Journal of Molecular and Cellular Virology . 2009,第1a2期

机译：SARS冠状病毒刺突蛋白诱导的先天免疫应答是通过体外人单核巨噬细胞中NF-κB途径的激活而发生的。
4. Protective Role of N-Acetyl-L-Tryptophan against Hepatic Ischemia-Reperfusion Injury by the TLR4/NF-KB Signaling pathway [C] . Jianxin Wang, Shuna Yu, Huiting Li, International Conference on Frontiers of Biological Sciences and Engineering . 2019

机译：N-乙酰-1-色氨酸对TLR4 / NF-KB信号通路对肝缺血再灌注损伤的保护作用
5. Role of the Vaccinia Virus E3 protein and its poxvirus orthologues in suppressing innate immune responses activated by RNA-based pathogen-associated molecular patterns. [D] . Myskiw, Chad. 2012

机译：牛痘病毒E3蛋白及其痘病毒直向同源物在抑制先天免疫应答中的作用，该先天免疫应答是由基于RNA的病原体相关分子模式激活的。
6. Donor Hepatic Steatosis Induce Exacerbated Ischemia-Reperfusion Injury through Activation of Innate Immune Response Molecular Pathways [O] . Ricardo C. Gehrau, Valeria R. Mas, Catherine I. Dumur, -1

机译：供体肝脂肪变性通过激活先天免疫反应分子途径诱导加重缺血再灌注损伤。
7. Donor Hepatic Steatosis Induce Exacerbated Ischemia-Reperfusion Injury Through Activation of Innate Immune Response Molecular Pathways [O] . Ricardo C. Gehrau, Valeria R. Mas, Catherine I. Dumur, 2015

机译：供体肝脏脂肪变性通过激活先天免疫应答分子途径诱导加剧缺血再灌注损伤

Donor Hepatic Steatosis Induce Exacerbated Ischemia-Reperfusion Injury Through Activation of Innate Immune Response Molecular Pathways

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