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首页> 外文期刊>Brain research >Corrigendum to 'Hydrocephalus and abnormal subcommissural organ in mice lacking presenilin-1 in Wnt1 cell lineages'(Brain Res. 1382 (2011) 275-281)
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Corrigendum to 'Hydrocephalus and abnormal subcommissural organ in mice lacking presenilin-1 in Wnt1 cell lineages'(Brain Res. 1382 (2011) 275-281)

机译:对“ Wnt1细胞谱系中缺乏早老素-1的小鼠的脑积水和异常的子下器官的勘误”(Brain Res。1382(2011)275-281)

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摘要

In this paper, we failed to cite appropriate references in several places. Revised text in the Discussion is as follows: 1) Among the causes of hydrocephalus are the overproduction of CSF by the choroid plexus and failure to drain the CSF at the subarachnoid space. Furthermore, blockage of CSF flow through the narrow Sylvian aqueduct is believed to be the primary cause of congenital hydrocephalus (Perez-Figares et al., 2001; Huh et al., 2009). 2) The SCO and the ventricular ependymal cells are thought to facilitate the flow of CSF through confining ventricular canals (Rodriguez et al., 1998; Meiniel, 2007; Huh et al., 2009).
机译:在本文中,我们未能在多个地方引用适当的参考文献。讨论中的修订文本如下:1)脑积水的原因包括脉络丛过度生产CSF和未能在蛛网膜下腔排空CSF。此外,脑脊液流过狭窄的Sylvian渡槽的阻塞被认为是先天性脑积水的主要原因(Perez-Figares等,2001; Huh等,2009)。 2)SCO和室间隔膜细胞被认为有助于脑脊液通过狭窄的心室管腔流动(Rodriguez等,1998; Meiniel,2007; Huh等,2009)。

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