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Labor and fetal heart rate decelerations: relation to fetal metabolic acidosis.

机译:劳动和胎儿心率减慢:与胎儿代谢性酸中毒有关。

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摘要

Although intrauterine fetal compromise during labor accounts for a small proportion of cerebral palsy cases, fetal hypoxic-ischemic encephalopathy remains an important and sometimes preventable etiology. This paper reviews the mechanisms regulating fetal heart rate responses, pathogenesis of fetal metabolic acidosis, and thresholds for metabolic acidosis associated with hypoxic-ischemic encephalopathy. The knowledge of normal changes in fetal metabolic acidosis during labor together with changes in association with fetal heart rate decelerations, can aid in the interpretation and management of fetal heart rate tracings.
机译:尽管分娩时宫内胎儿损害占脑瘫病例的一小部分,但胎儿缺氧缺血性脑病仍然是重要的病因,有时是可预防的。本文综述了调节胎儿心率反应的机制,胎儿代谢性酸中毒的发病机制以及与缺氧缺血性脑病相关的代谢性酸中毒的阈值。对分娩过程中胎儿代谢性酸中毒的正常变化以及与胎儿心率减速相关的变化的了解,可以帮助解释和管理胎儿心率描记。

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