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An approach to noncavotricuspid isthmus dependent flutter.

机译:一种非腔窦峡部依赖扑动的方法。

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The archetypical atrial macro-reentrant atrial arrhythmia is cavotricuspid isthmus dependent counterclockwise flutter. This arrhythmia is bounded posteriorly by the crista terminalisand anteriorly by the tricuspid annulus, and uses the cavotricuspid isthmus as a critical zone of slowed conduction that supports the reentrant mechanism. These features, together with recognition of the characteristic surface electrocardiogram, have served to facilitate the rapid recognition and evolution of a highly successful ablation strategy for the cure of this arrhythmia. Furthermore, electrocardiographicand mapping studies have identified that this isthmus of conduction could support such macro-reentry in either orientation, which could also be cured by the same ablation strategy. Increasingly, such macro-reentry can occur in any region of the atria in the presence of adequate conduction barriers that support the substrate for reentry, manifesting as sustained monornorphic regular atrial tachycardias that are occasionally referred to as type II or atypical flutters. These are noncavotricuspid isthmus dependent flutters and may occur in either the right, left, or both atria in patients with or without underlying structural heart disease. While such macro-reentrant flutters frequently develop following previous cardiac surgery or ablation (particularly of atrial fibrillation), they have also been observed to occur in the absence of prior intervention when arrhythmia is supported by spontaneous regions of conduction abnormalities or electrical silence. This article will focus on the clinical approach used for the mapping and ablation of atypical flutters at Hopital Cardologique du Haut-Leveque and the Universite Victor Segalen Bordeaux II, Bordeaux, France.
机译:原型房性大折返性房性心律失常是依赖于左室窦峡部逆时针扑动。这种心律失常在后部由cr末端界定,在前部由三尖瓣环界定,并使用左房窦峡部作为支持再入机制的慢传导的关键区域。这些特征与特征性表面心电图的识别一起,有助于快速识别并成功开发出一种非常成功的消融策略,以治愈这种心律不齐。此外,心电图和作图研究已经确定,这种传导峡部可以在任一方向上支持这种巨大的折返,也可以通过相同的消融策略治愈。在有足够的传导屏障支持基底再进入的情况下,这种大范围再进入可在心房的任何区域中发生,表现为持续的单发性规则性房​​性心动过速,有时被称为II型或非典型扑动。这些是非腔窦性峡部依赖性扑动,可能发生在有或没有基础结构性心脏病的患者的右,左或两个心房。虽然这种大型凹入式扑动在先前的心脏手术或消融后(尤其是房颤)经常发生,但也可以观察到,当心律失常由传导异常或电沉默的自发性区域支持时,在没有事先干预的情况下就会发生。本文将重点讨论用于在上洛韦克心脏医院心脏跳动和法国波尔多维克多·塞加仑波尔多二世大学进行非典型扑扑标测和消融的临床方法。

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